Publications

Found 7 results
Filters: Author is Y.Q. Yang  [Clear All Filters]
2015
Y. Q. Yang, Wang, H., Liang, M. L., Yan, J. L., Liu, L., Li, C. Y., and Yang, J., “Construction and expression of prokaryotic expression vectors fused with genes of Magnaporthe oryzae effector proteins and mCherry”, vol. 14, pp. 10827-10836, 2015.
M. L. Liang, Yan, J. L., Yang, Y. Q., Liu, L., Li, C. Y., and Yang, J., “Construction of overexpression vectors of Magnaporthe oryzae genes BAS1 and BAS4 fusion to mCherry and screening of overexpression strains”, vol. 14, pp. 7068-7078, 2015.
J. Yang, Liang, M. L., Yan, J. L., Yang, Y. Q., Liu, L., Liu, C., Yang, L. J., and Li, C. Y., “Expression of Magnaporthe oryzae genes encoding cysteine-rich proteins secreted during nitrogen starvation and interaction with its host, Oryza sativa”, vol. 14, pp. 17099-17108, 2015.
J. Z. Peng, Xue, L., Chen, J., Chen, B. S., and Yang, Y. Q., “Influence of cyclophilin D protein expression level on endothelial cell oxidative damage resistance”, vol. 14, pp. 4258-4268, 2015.
R. Li, Wang, B., He, C. Q., Yang, Y. Q., Guo, H., Chen, Y., and Du, T. H., “Upregulation of fibroblast growth factor 1 in the synovial membranes of patients with late stage osteoarthritis”, vol. 14, pp. 11191-11199, 2015.
2013
Y. T. Hui, Yang, Y. Q., Liu, R. Y., Zhang, Y. Y., Xiang, C. J., Liu, Z. Z., Ding, Y. H., Zhang, Y. L., and Wang, B. R., “Significant association of APOA5 and APOC3 gene polymorphisms with meat quality traits in Kele pigs”, vol. 12, pp. 3643-3650, 2013.
2011
J. Wang, Liu, X. Y., and Yang, Y. Q., “Novel NKX2-5 mutations responsible for congenital heart disease”, vol. 10, pp. 2905-2915, 2011.
Akazawa H and Komuro I (2005). Cardiac transcription factor Csx/Nkx2-5: its role in cardiac development and diseases. Pharmacol. Ther. 107: 252-268. http://dx.doi.org/10.1016/j.pharmthera.2005.03.005 Benson DW, Silberbach GM, Kavanaugh-McHugh A, Cottrill C, et al. (1999). Mutations in the cardiac transcription factor NKX2.5 affect diverse cardiac developmental pathways. J. Clin. Invest. 104: 1567-1573. http://dx.doi.org/10.1172/JCI8154 PMid:10587520    PMCid:409866 Biben C, Weber R, Kesteven S, Stanley E, et al. (2000). Cardiac septal and valvular dysmorphogenesis in mice heterozygous for mutations in the homeobox gene Nkx2-5. Circ. Res. 87: 888-895. PMid:11073884 Briggs LE, Takeda M, Cuadra AE, Wakimoto H, et al. (2008). Perinatal loss of Nkx2-5 results in rapid conduction and contraction defects. Circ. Res. 103: 580-590. http://dx.doi.org/10.1161/CIRCRESAHA.108.171835 PMid:18689573    PMCid:2590500 Ching YH, Ghosh TK, Cross SJ, Packham EA, et al. (2005). Mutation in myosin heavy chain 6 causes atrial septal defect. Nat. Genet. 37: 423-428. http://dx.doi.org/10.1038/ng1526 PMid:15735645 Elliott DA, Kirk EP, Yeoh T, Chandar S, et al. (2003). Cardiac homeobox gene NKX2-5 mutations and congenital heart disease: associations with atrial septal defect and hypoplastic left heart syndrome. J. Am. Coll. Cardiol. 41: 2072-2076. http://dx.doi.org/10.1016/S0735-1097(03)00420-0 Garg V, Kathiriya IS, Barnes R, Schluterman MK, et al. (2003). GATA4 mutations cause human congenital heart defects and reveal an interaction with TBX5. Nature 424: 443-447. http://dx.doi.org/10.1038/nature01827 PMid:12845333 Garg V, Muth AN, Ransom JF, Schluterman MK, et al. (2005). Mutations in NOTCH1 cause aortic valve disease. Nature 437: 270-274. http://dx.doi.org/10.1038/nature03940 PMid:16025100 Grow MW and Krieg PA (1998). Tinman function is essential for vertebrate heart development: elimination of cardiac differentiation by dominant inhibitory mutants of the tinman-related genes, XNkx2-3 and XNkx2-5. Dev. Biol. 204: 187-196. http://dx.doi.org/10.1006/dbio.1998.9080 PMid:9851852 Hirayama-Yamada K, Kamisago M, Akimoto K, Aotsuka H, et al. (2005). Phenotypes with GATA4 or NKX2.5 mutations in familial atrial septal defect. Am. J. Med. Genet. A 135: 47-52. http://dx.doi.org/10.1002/ajmg.a.30684 PMid:15810002 Hosoda T, Komuro I, Shiojima I, Hiroi Y, et al. (1999). Familial atrial septal defect and atrioventricular conduction disturbance associated with a point mutation in the cardiac homeobox gene CSX/NKX2-5 in a Japanese patient. Jpn. Circ. J. 63: 425-426. http://dx.doi.org/10.1253/jcj.63.425 PMid:10943630 Jenkins KJ, Correa A, Feinstein JA, Botto L, et al. (2007). Noninherited risk factors and congenital cardiovascular defects: current knowledge: a scientific statement from the American Heart Association Council on Cardiovascular Disease in the Young: endorsed by the American Academy of Pediatrics. Circulation 115: 2995-3014. http://dx.doi.org/10.1161/CIRCULATIONAHA.106.183216 PMid:17519397 Kasahara H and Benson DW (2004). Biochemical analyses of eight NKX2.5 homeodomain missense mutations causing atrioventricular block and cardiac anomalies. Cardiovasc. Res. 64: 40-51. http://dx.doi.org/10.1016/j.cardiores.2004.06.004 PMid:15364612 Kasahara H, Lee B, Schott JJ, Benson DW, et al. (2000). Loss of function and inhibitory effects of human CSX/NKX2.5 homeoprotein mutations associated with congenital heart disease. J. Clin. Invest. 106: 299-308. http://dx.doi.org/10.1172/JCI9860 PMid:10903346    PMCid:314312 Kasahara H, Wakimoto H, Liu M, Maguire CT, et al. (2001). Progressive atrioventricular conduction defects and heart failure in mice expressing a mutant Csx/Nkx2.5 homeoprotein. J. Clin. Invest. 108: 189-201. PMid:11457872    PMCid:203028 Kodo K, Nishizawa T, Furutani M, Arai S, et al. (2009). GATA6 mutations cause human cardiac outflow tract defects by disrupting semaphorin-plexin signaling. Proc Natl. Acad. Sci U. S. A. 106: 13933-13938. http://dx.doi.org/10.1073/pnas.0904744106 PMid:19666519    PMCid:2728998 Li QY, Newbury-Ecob RA, Terrett JA, Wilson DI, et al. (1997). Holt-Oram syndrome is caused by mutations in TBX5, a member of the Brachyury (T) gene family. Nat. Genet. 15: 21-29. http://dx.doi.org/10.1038/ng0197-21 PMid:8988164 Lloyd-Jones D, Adams R, Carnethon M, De Simone G, et al. (2009). Heart disease and stroke statistics--2009 update: a report from the American Heart Association Statistics Committee and Stroke Statistics Subcommittee. Circulation 119: e21-181. http://dx.doi.org/10.1161/CIRCULATIONAHA.108.191261 PMid:19075105 Lyons I, Parsons LM, Hartley L, Li R, et al. (1995). Myogenic and morphogenetic defects in the heart tubes of murine embryos lacking the homeo box gene Nkx2-5. Genes Dev. 9: 1654-1666. http://dx.doi.org/10.1101/gad.9.13.1654 Matsson H, Eason J, Bookwalter CS, Klar J, et al. (2008). Alpha-cardiac actin mutations produce atrial septal defects. Hum. Mol. Genet. 17: 256-265. http://dx.doi.org/10.1093/hmg/ddm302 PMid:17947298 Nora JJ and Nora AH (1976). Recurrence risks in children having one parent with a congenital heart disease. Circulation 53: 701-702. PMid:1253394 Pashmforoush M, Lu JT, Chen H, Amand TS, et al. (2004). Nkx2-5 pathways and congenital heart disease; loss of ventricular myocyte lineage specification leads to progressive cardiomyopathy and complete heart block. Cell 117: 373-386. http://dx.doi.org/10.1016/S0092-8674(04)00405-2 Pierpont ME, Basson CT, Benson DW Jr, Gelb BD, et al. (2007). Genetic basis for congenital heart defects: current knowledge: a scientific statement from the American Heart Association Congenital Cardiac Defects Committee, Council on Cardiovascular Disease in the Young: endorsed by the American Academy of Pediatrics. Circulation 115: 3015-3038. http://dx.doi.org/10.1161/CIRCULATIONAHA.106.183056 PMid:17519398 Prall OW, Menon MK, Solloway MJ, Watanabe Y, et al. (2007). An Nkx2-5/Bmp2/Smad1 negative feedback loop controls heart progenitor specification and proliferation. Cell 128: 947-959. http://dx.doi.org/10.1016/j.cell.2007.01.042 PMid:17350578    PMCid:2092439 Razzaque MA, Nishizawa T, Komoike Y, Yagi H, et al. (2007). Germline gain-of-function mutations in RAF1 cause Noonan syndrome. Nat. Genet. 39: 1013-1017. http://dx.doi.org/10.1038/ng2078 PMid:17603482 Reamon-Buettner SM and Borlak J (2004). Somatic NKX2-5 mutations as a novel mechanism of disease in complex congenital heart disease. J. Med. Genet. 41: 684-690. http://dx.doi.org/10.1136/jmg.2003.017483 PMid:15342699    PMCid:1735891 Roberts AE, Araki T, Swanson KD, Montgomery KT, et al. (2007). Germline gain-of-function mutations in SOS1 cause Noonan syndrome. Nat. Genet. 39: 70-74. http://dx.doi.org/10.1038/ng1926 PMid:17143285 Rodriguez-Viciana P, Tetsu O, Tidyman WE, Estep AL, et al. (2006). Germline mutations in genes within the MAPK pathway cause cardio-facio-cutaneous syndrome. Science 311: 1287-1290. http://dx.doi.org/10.1126/science.1124642 PMid:16439621 Sarkozy A, Conti E, Neri C, D’Agostino R, et al. (2005). Spectrum of atrial septal defects associated with mutations of NKX2.5 and GATA4 transcription factors. J. Med. Genet. 42: e16. http://dx.doi.org/10.1136/jmg.2004.026740 PMid:15689439    PMCid:1735979 Schott JJ, Benson DW, Basson CT, Pease W, et al. (1998). Congenital heart disease caused by mutations in the transcription factor NKX2-5. Science 281: 108-111. http://dx.doi.org/10.1126/science.281.5373.108 PMid:9651244 Shiojima I, Komuro I, Inazawa J, Nakahori Y, et al. (1995). Assignment of cardiac homeobox gene CSX to human chromosome 5q34. Genomics 27: 204-206. http://dx.doi.org/10.1006/geno.1995.1027 PMid:7665173 Stallmeyer B, Fenge H, Nowak-Gottl U and Schulze-Bahr E (2010). Mutational spectrum in the cardiac transcription factor gene NKX2.5 (CSX) associated with congenital heart disease. Clin. Genet. 78: 533-540. http://dx.doi.org/10.1111/j.1399-0004.2010.01422.x PMid:20456451 Tanaka M, Chen Z, Bartunkova S, Yamasaki N, et al. (1999). The cardiac homeobox gene Csx/Nkx2.5 lies genetically upstream of multiple genes essential for heart development. Development 126: 1269-1280. PMid:10021345