Publications
Found 13 results
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“Gene expression profile in breast cancer comprising predictive markers for metastatic risk”, vol. 14, pp. 10929-10936, 2015.
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“Dithranol downregulates expression of Id1 mRNA in human keratinocytes in vitro”, vol. 11, pp. 3290-3297, 2012.
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Afifi T, de GG, Huang C and Zhou Y (2005). Topical therapies for psoriasis: evidence-based review. Can. Fam. Physician 51: 519-525.
PMid:15856971 PMCid:1472956
Benezra R, Davis RL, Lockshon D, Turner DL, et al. (1990). The protein Id: a negative regulator of helix-loop-helix DNA binding proteins. Cell 61: 49-59.
http://dx.doi.org/10.1016/0092-8674(90)90214-Y
Bjorntorp E, Parsa R, Thornemo M, Wennberg AM, et al. (2003). The helix-loop-helix transcription factor Id1 is highly expressed in psoriatic involved skin. Acta Derm. Venereol. 83: 403-409.
http://dx.doi.org/10.1080/00015550310015806
PMid:14690332
Boukamp P, Petrussevska RT, Breitkreutz D, Hornung J, et al. (1988). Normal keratinization in a spontaneously immortalized aneuploid human keratinocyte cell line. J. Cell. Biol. 106: 761-771.
http://dx.doi.org/10.1083/jcb.106.3.761
PMid:2450098
Bradford MM (1976). A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. Anal. Biochem. 72: 248-254.
http://dx.doi.org/10.1016/0003-2697(76)90527-3
Cheng YJ, Tsai JW, Hsieh KC, Yang YC, et al. (2011). Id1 promotes lung cancer cell proliferation and tumor growth through Akt-related pathway. Cancer Lett. 307: 191-199.
http://dx.doi.org/10.1016/j.canlet.2011.04.003
PMid:21536374
Farkas A, Kemeny L, Szony BJ, Bata-Csorgo Z, et al. (2001). Dithranol upregulates IL-10 receptors on the cultured human keratinocyte cell line HaCaT. Inflamm. Res. 50: 44-49.
http://dx.doi.org/10.1007/s000110050723
PMid:11235021
George SE, Anderson RJ, Cunningham A, Donaldson M, et al. (2010). Evaluation of a range of anti-proliferative assays for the preclinical screening of anti-psoriatic drugs: a comparison of colorimetric and fluorimetric assays with the thymidine incorporation assay. Assay Drug Dev. Technol. 8: 389-400.
http://dx.doi.org/10.1089/adt.2009.0224
PMid:20482335
Gry M, Rimini R, Stromberg S, Asplund A, et al. (2009). Correlations between RNA and protein expression profiles in 23 human cell lines. BMC Genomics 10: 365.
http://dx.doi.org/10.1186/1471-2164-10-365
PMid:19660143 PMCid:2728742
Guo Y, Xiao P, Lei S, Deng F, et al. (2008). How is mRNA expression predictive for protein expression? A correlation study on human circulating monocytes. Acta Biochim. Biophys. Sin. 40: 426-436.
http://dx.doi.org/10.1111/j.1745-7270.2008.00418.x
Hamajima Y, Komori M, Preciado DA, Choo DI, et al. (2010). The role of inhibitor of DNA-binding (Id1) in hyperproliferation of keratinocytes: the pathological basis for middle ear cholesteatoma from chronic otitis media. Cell Prolif. 43: 457-463.
http://dx.doi.org/10.1111/j.1365-2184.2010.00695.x
PMid:20887552 PMCid:2950310
Jacobson CC, Kumar S and Kimball AB (2011). Latitude and psoriasis prevalence. J. Am. Acad. Dermatol. 65: 870-873.
http://dx.doi.org/10.1016/j.jaad.2009.05.047
PMid:21920244
Kamili QU and Menter A (2009). Topical treatment of psoriasis. Curr. Probl. Dermatol. 38: 37-58.
http://dx.doi.org/10.1159/000232303
PMid:19710549
Lichtinghagen R, Musholt PB, Lein M, Römer A, et al. (2002). Different mRNA and protein expression of matrix metalloproteinases 2 and 9 and tissue inhibitor of metalloproteinases 1 in benign and malignant prostate tissue. Eur. Urol. 42: 398-406.
http://dx.doi.org/10.1016/S0302-2838(02)00324-X
Ling MT, Lau TC, Zhou C, Chua CW, et al. (2005). Overexpression of Id-1 in prostate cancer cells promotes angiogenesis through the activation of vascular endothelial growth factor (VEGF). Carcinogenesis 26: 1668-1676.
http://dx.doi.org/10.1093/carcin/bgi128
PMid:15905202
Ling YX, Tao J, Fang SF, Hui Z, et al. (2011). Downregulation of Id1 by small interfering RNA in prostate cancer PC3 cells in vivo and in vitro. Eur. J. Cancer Prev. 20: 9-17.
http://dx.doi.org/10.1097/CEJ.0b013e32833ebaa0
PMid:20881502
Lister J, Forrester WC and Baron MH (1995). Inhibition of an erythroid differentiation switch by the helix-loop-helix protein Id1. J. Biol. Chem. 270: 17939-17946.
http://dx.doi.org/10.1074/jbc.270.30.17939
PMid:7629100
Lyden D, Young AZ, Zagzag D, Yan W, et al. (1999). Id1 and Id3 are required for neurogenesis, angiogenesis and vascularization of tumour xenografts. Nature 401: 670-677.
http://dx.doi.org/10.1038/44334
PMid:10537105
Mark EB, Jonsson M, Asp J, Wennberg AM, et al. (2006). Expression of genes involved in the regulation of p16 in psoriatic involved skin. Arch. Dermatol. Res. 297: 459-467.
http://dx.doi.org/10.1007/s00403-006-0649-1
PMid:16552541
Massari ME and Murre C (2000). Helix-loop-helix proteins: regulators of transcription in eucaryotic organisms. Mol. Cell Biol. 20: 429-440.
http://dx.doi.org/10.1128/MCB.20.2.429-440.2000
PMid:10611221 PMCid:85097
McAllister SD, Christian RT, Horowitz MP, Garcia A, et al. (2007). Cannabidiol as a novel inhibitor of Id-1 gene expression in aggressive breast cancer cells. Mol. Cancer Ther. 6: 2921-2927.
http://dx.doi.org/10.1158/1535-7163.MCT-07-0371
PMid:18025276
McAllister SD, Murase R, Christian RT, Lau D, et al. (2011). Pathways mediating the effects of cannabidiol on the reduction of breast cancer cell proliferation, invasion, and metastasis. Breast Cancer Res. Treat. 129: 37-47.
http://dx.doi.org/10.1007/s10549-010-1177-4
PMid:20859676 PMCid:3410650
Ouyang XS, Wang X, Lee DT, Tsao SW, et al. (2002). Over expression of ID-1 in prostate cancer. J. Urol. 167: 2598-2602.
http://dx.doi.org/10.1016/S0022-5347(05)65044-6
Pavithran K (2001). Psoriasis: topical treatment. Indian J. Dermatol. Venereol. Leprol. 67: 85.
PMid:17664716
Ristow HJ (1996). Studies on stimulation of DNA synthesis with epidermal growth factor and insulin-like growth factor-I in cultured human keratinocytes. Growth Regul. 6: 96-109.
PMid:8781986
Ronpirin C and Tencomnao T (2012). Effects of the antipsoriatic drug dithranol on E2A and caspase-9 gene expression in vitro. Genet. Mol. Res. 11: 412-420.
http://dx.doi.org/10.4238/2012.February.17.3
PMid:22370944
Ronpirin C, Achariyakul M, Tencomnao T, Wongpiyabovorn J, et al. (2010). Up-regulation of Id1 in peripheral blood of psoriatic patients. Genet. Mol. Res. 9: 2239-2247.
http://dx.doi.org/10.4238/vol9-4gmr963
PMid:21086260
Saelee C, Thongrakard V and Tencomnao T (2011). Effects of Thai medicinal herb extracts with anti-psoriatic activity on the expression on NF-kappaB signaling biomarkers in HaCaT keratinocytes. Molecules 16: 3908-3932.
http://dx.doi.org/10.3390/molecules16053908
PMid:21555979
Sakurai D, Yamaguchi A, Tsuchiya N, Yamamoto K, et al. (2001). Expression of ID family genes in the synovia from patients with rheumatoid arthritis. Biochem. Biophys. Res. Commun. 284: 436-442.
http://dx.doi.org/10.1006/bbrc.2001.4974
PMid:11394898
Schindl M, Schoppmann SF, Strobel T, Heinzl H, et al. (2003). Level of Id-1 protein expression correlates with poor differentiation, enhanced malignant potential, and more aggressive clinical behavior of epithelial ovarian tumors. Clin. Cancer Res. 9: 779-785.
PMid:12576450
Schoppmann SF, Schindl M, Bayer G, Aumayr K, et al. (2003). Overexpression of Id-1 is associated with poor clinical outcome in node negative breast cancer. Int. J. Cancer 104: 677-682.
http://dx.doi.org/10.1002/ijc.11009
PMid:12640673
Tencomnao T, Ronpirin C, Prasansuklab A and Poovorawan Y (2009). Decreased EGFR mRNA expression in response to antipsoriatic drug dithranol in vitro. Afr. J. Biotechnol. 8: 3141-3146.
Villano CM and White LA (2006). Expression of the helix-loop-helix protein inhibitor of DNA binding-1 (ID-1) is activated by all-trans retinoic acid in normal human keratinocytes. Toxicol. Appl. Pharmacol. 214: 219-229.
http://dx.doi.org/10.1016/j.taap.2005.12.015
PMid:16494909
Wong YC, Wang X and Ling MT (2004). Id-1 expression and cell survival. Apoptosis 9: 279-289.
http://dx.doi.org/10.1023/B:APPT.0000025804.25396.79
PMid:15258459
“Effects of the antipsoriatic drug dithranol on E2A and caspase-9 gene expression in vitro”, vol. 11, pp. 412-420, 2012.
, Allen D, Winters E, Kenna PF, Humphries P, et al. (2008). Reference gene selection for real-time rtPCR in human epidermal keratinocytes. J. Dermatol. Sci. 49: 217-225.
http://dx.doi.org/10.1016/j.jdermsci.2007.10.001
PMid:18061409
Bjorntorp E, Parsa R, Thornemo M, Wennberg AM, et al. (2003). The helix-loop-helix transcription factor Id1 is highly expressed in psoriatic involved skin. Acta Derm. Venereol. 83: 403-409.
http://dx.doi.org/10.1080/00015550310015806
PMid:14690332
Boukamp P, Petrussevska RT, Breitkreutz D, Hornung J, et al. (1988). Normal keratinization in a spontaneously immortalized aneuploid human keratinocyte cell line. J. Cell Biol. 106: 761-771.
http://dx.doi.org/10.1083/jcb.106.3.761
PMid:2450098
Bradford MM (1976). A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. Anal. Biochem. 72: 248-254.
http://dx.doi.org/10.1016/0003-2697(76)90527-3
Engel I and Murre C (2001). The function of E- and Id proteins in lymphocyte development. Nat. Rev. Immunol. 1: 193- 199.
http://dx.doi.org/10.1038/35105060
PMid:11905828
Farkas A, Kemeny L, Szony BJ, Bata-Csorgo Z, et al. (2001). Dithranol upregulates IL-10 receptors on the cultured human keratinocyte cell line HaCaT. Inflamm. Res. 50: 44-49.
http://dx.doi.org/10.1007/s000110050723
PMid:11235021
Henseleit U, Rosenbach T and Kolde G (1996). Induction of apoptosis in human HaCaT keratinocytes. Arch. Dermatol. Res. 288: 676-683.
http://dx.doi.org/10.1007/BF02505277
PMid:8931870
Kee BL (2009). E and ID proteins branch out. Nat. Rev. Immunol. 9: 175-184.
http://dx.doi.org/10.1038/nri2507
PMid:19240756
Kemeny L, Ruzicka T and Braun-Falco O (1990). Dithranol: a review of the mechanism of action in the treatment of psoriasis vulgaris. Skin Pharmacol. 3: 1-20.
http://dx.doi.org/10.1159/000210836
Kuida K (2000). Caspase-9. Int. J. Biochem. Cell Biol. 32: 121-124.
http://dx.doi.org/10.1016/S1357-2725(99)00024-2
Langlands K, Down GA and Kealey T (2000). Id proteins are dynamically expressed in normal epidermis and dysregulated in squamous cell carcinoma. Cancer Res. 60: 5929-5933.
PMid:11085505
Lowes MA, Bowcock AM and Krueger JG (2007). Pathogenesis and therapy of psoriasis. Nature 445: 866-873.
http://dx.doi.org/10.1038/nature05663
PMid:17314973
Mark EB, Jonsson M, Asp J, Wennberg AM, et al. (2006). Expression of genes involved in the regulation of p16 in psoriatic involved skin. Arch. Dermatol. Res. 297: 459-467.
http://dx.doi.org/10.1007/s00403-006-0649-1
PMid:16552541
Olson EN and Klein WH (1994). bHLH factors in muscle development: dead lines and commitments, what to leave in and what to leave out. Genes Dev. 8: 1-8.
http://dx.doi.org/10.1101/gad.8.1.1
Oztas P, Lortlar N, Oztas MO, Omeroglu S, et al. (2006). Caspase 9 is decreased in psoriatic epidermis. Acta Histochem. 108: 497-499.
http://dx.doi.org/10.1016/j.acthis.2006.09.002
PMid:17034836
Pavithran K (2001). Psoriasis: topical treatment. Indian J. Dermatol. Venereol. Leprol. 67: 85.
PMid:17664716
Perk J, Iavarone A and Benezra R (2005). Id family of helix-loop-helix proteins in cancer. Nat. Rev. Cancer 5: 603-614.
http://dx.doi.org/10.1038/nrc1673
PMid:16034366
Raymond AA, Mechin MC, Nachat R, Toulza E, et al. (2007). Nine procaspases are expressed in normal human epidermis, but only caspase-14 is fully processed. Br. J. Dermatol. 156: 420-427.
http://dx.doi.org/10.1111/j.1365-2133.2006.07656.x
PMid:17300228
Ronpirin C, Achariyakul M, Tencomnao T, Wongpiyabovorn J, et al. (2010). Up-regulation of Id1 in peripheral blood of psoriatic patients. Genet. Mol. Res. 9: 2239-2247.
http://dx.doi.org/10.4238/vol9-4gmr963
PMid:21086260
Schon MP and Boehncke WH (2005). Psoriasis. N. Engl. J. Med. 352: 1899-1912.
http://dx.doi.org/10.1056/NEJMra041320
PMid:15872205
Tencomnao T, Ronpirin C, Prasansuklab A and Poovorawan Y (2009). Decreased EGFR mRNA expression in response to antipsoriatic drug dithranol in vitro. Afr. J. Biotechnol. 8: 3141-3146.
Yamamoto T and Nishioka K (2003). Alteration of the expression of Bcl-2, Bcl-x, Bax, Fas, and Fas ligand in the involved skin of psoriasis vulgaris following topical anthralin therapy. Skin Pharmacol. Appl. Skin Physiol. 16: 50-58.
http://dx.doi.org/10.1159/000068289
Yan W, Young AZ, Soares VC, Kelley R, et al. (1997). High incidence of T-cell tumors in E2A-null mice and E2A/Id1 double-knockout mice. Mol. Cell Biol. 17: 7317-7327.
PMid:9372963 PMCid:232588
“Inhibitory effect of alternatively spliced RAGEv1 on the expression of NF-kB and TNF-α in hepatocellular carcinoma cells”, vol. 11, pp. 1712-1720, 2012.
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Aggarwal BB and Shishodia S (2006). Molecular targets of dietary agents for prevention and therapy of cancer. Biochem. Pharmacol. 71: 1397-1421.
http://dx.doi.org/10.1016/j.bcp.2006.02.009
PMid:16563357
Ahmed N (2005). Advanced glycation endproducts-role in pathology of diabetic complications. Diabetes Res. Clin. Pract. 67: 3-21.
http://dx.doi.org/10.1016/j.diabres.2004.09.004
PMid:15620429
Emanuele E, D'Angelo A, Tomaino C, Binetti G, et al. (2005). Circulating levels of soluble receptor for advanced glycation end products in Alzheimer disease and vascular dementia. Arch. Neurol. 62: 1734-1736.
http://dx.doi.org/10.1001/archneur.62.11.1734
PMid:16286548
Falcone C, Emanuele E, D'Angelo A, Buzzi MP, et al. (2005). Plasma levels of soluble receptor for advanced glycation end products and coronary artery disease in nondiabetic men. Arterioscler. Thromb. Vasc. Biol. 25: 1032-1037.
http://dx.doi.org/10.1161/01.ATV.0000160342.20342.00
PMid:15731496
Fernandez-Botran R, Crespo FA and Sun X (2002). Soluble cytokine receptors in biological therapy. Expert. Opin. Biol. Ther. 2: 585-605.
http://dx.doi.org/10.1517/14712598.2.6.585
PMid:12171504
Fisker S, Hansen B, Fuglsang J, Kristensen K, et al. (2004). Gene expression of the GH receptor in subcutaneous and intraabdominal fat in healthy females: relationship to GH-binding protein. Eur. J. Endocrinol. 150: 773-777.
http://dx.doi.org/10.1530/eje.0.1500773
PMid:15191346
Geroldi D, Falcone C, Emanuele E, D'Angelo A, et al. (2005). Decreased plasma levels of soluble receptor for advanced glycation end-products in patients with essential hypertension. J. Hypertens. 23: 1725-1729.
http://dx.doi.org/10.1097/01.hjh.0000177535.45785.64
PMid:16093918
Hirata K, Takada M, Suzuki Y and Kuroda Y (2003). Expression of receptor for advanced glycation end products (RAGE) in human biliary cancer cells. Hepatogastroenterology 50: 1205-1207.
PMid:14571699
Hiwatashi K, Ueno S, Abeyama K, Kubo F, et al. (2008). A novel function of the receptor for advanced glycation end-products (RAGE) in association with tumorigenesis and tumor differentiation of HCC. Ann. Surg. Oncol. 15: 923-933.
http://dx.doi.org/10.1245/s10434-007-9698-8
PMid:18080716 PMCid:2234441
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Hudson BI, Carter AM, Harja E, Kalea AZ, et al. (2008). Identification, classification, and expression of RAGE gene splice variants. FASEB J. 22: 1572-1580.
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“Semi-quantitative detection of gene expression using bisbenzimide dye”, vol. 10. pp. 3747-3759, 2011.
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Ahmed MU, Idegami K, Chikae M, Kerman K, et al. (2007). Electrochemical DNA biosensor using a disposable electrochemical printed (DEP) chip for the detection of SNPs from unpurified PCR amplicons. Analyst 132: 431-438.
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Fisker S, Hansen B, Fuglsang J, Kristensen K, et al. (2004). Gene expression of the GH receptor in subcutaneous and intraabdominal fat in healthy females: relationship to GH-binding protein. Eur. J. Endocrinol. 150: 773-777.
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Hudson BI, Carter AM, Harja E, Kalea AZ, et al. (2008). Identification, classification, and expression of RAGE gene splice variants. FASEB J. 22: 1572-1580.
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Kobayashi M, Kusakawa T, Saito M, Kaji S, et al. (2004). Electrochemical DNA quantification based on aggregation induced by Hoechst 33258. Electrochem. Commun. 6: 337-343.
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Lertwittayapon T (2010). Cloning of Soluble Receptor for Advanced Glycation Endproducts (sRAGE) Gene and Study the Effect of its Expressed Product on Liver Cancer Cell. Master's thesis. Chulalongkorn Uninversity, Bangkok.
Nagaev I and Smith U (2001). Insulin resistance and type 2 diabetes are not related to resistin expression in human fat cells or skeletal muscle. Biochem. Biophys. Res. Commun. 285: 561-564.
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Schmidt AM, Yan SD, Yan SF and Stern DM (2001). The multiligand receptor RAGE as a progression factor amplifying immune and inflammatory responses. J. Clin. Invest. 108: 949-955.
PMid:11581294 PMCid:200958
Stern DM, Yan SD, Yan SF and Schmidt AM (2002). Receptor for advanced glycation endproducts (RAGE) and the complications of diabetes. Ageing Res. Rev. 1: 1-15.
http://dx.doi.org/10.1016/S0047-6374(01)00366-9
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