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“Lack of association of the G22A polymorphism of the ADA gene in patients with ankylosing spondylitis”, vol. 11, pp. 1178-1184, 2012.
, Bottini E, Carapella E, Cataldi L, Nicotra M, et al. (1981). Adenosine deaminase polymorphism. Associations at clinical level suggest a role in cell functions and immune reactions. J. Med. Genet. 18: 331-334.
http://dx.doi.org/10.1136/jmg.18.5.331
PMid:7199090 PMCid:1048752
Breban M (2006). Genetics of spondyloarthritis. Best Pract. Res. Clin. Rheumatol. 20: 593-599.
http://dx.doi.org/10.1016/j.berh.2006.03.002
PMid:16777584
Brionez TF and Reveille JD (2008). The contribution of genes outside the major histocompatibility complex to susceptibility to ankylosing spondylitis. Curr. Opin. Rheumatol. 20: 384-391.
http://dx.doi.org/10.1097/BOR.0b013e32830460fe
PMid:18525349
Dutra GP, Ottoni GL, Lara DR and Bogo MR (2010). Lower frequency of the low activity adenosine deaminase allelic variant (ADA1*2) in schizophrenic patients. Rev. Bras. Psiquiatr. 32: 275-278.
http://dx.doi.org/10.1590/S1516-44462010005000003
PMid:20414589
Goie HS, Steven MM, Van der Linden SM and Cats A (1985). Evaluation of diagnostic criteria for ankylosing spondylitis: a comparison of the Rome, New York and modified New York criteria in patients with a positive clinical history screening test for ankylosing spondylitis. Rheumatology 24: 242-249.
http://dx.doi.org/10.1093/rheumatology/24.3.242
Hirschhorn R, Yang DR and Israni A (1994). An Asp8Asn substitution results in the adenosine deaminase (ADA) genetic polymorphism (ADA 2 allozyme): occurrence on different chromosomal backgrounds and apparent intragenic crossover. Ann. Hum. Genet. 58: 1-9.
http://dx.doi.org/10.1111/j.1469-1809.1994.tb00720.x
PMid:8031011
Lima I, Néri F and Santiago MB (2005). Dosagem sérica de adenosina deaminase em lúpus eritematoso sistêmico: ausência de associação com atividade da doença. Rev. Bras. Reumatol. 45: 273-279.
http://dx.doi.org/10.1590/S0482-50042005000500002
Liu Y, Saccucci P, Qi H, Wu HC, et al. (2006). ADA polymorphisms and asthma: a study in the Chinese Han population. J. Asthma 43: 203-206.
http://dx.doi.org/10.1080/02770900600566827
PMid:16754522
Lopez de Castro JA (2007). HLA-B27 and the pathogenesis of spondyloarthropathies. Immunol. Lett. 108: 27-33.
http://dx.doi.org/10.1016/j.imlet.2006.10.004
PMid:17129613
Nunes DP, Spegiorin LC, de Mattos CC, Oliani AH, et al. (2011). The ADA*2 allele of the adenosine deaminase gene (20q13.11) and recurrent spontaneous abortions: an age-dependent association. Clinics 66: 1929-1933.
http://dx.doi.org/10.1590/S1807-59322011001100013
PMid:22086524 PMCid:3203966
Saccucci P, Manca Bitti ML, Bottini N, Rapini N, et al. (2009). Type 1 diabetes: evidence of interaction between ACP1 and ADA1 gene polymorphisms. Med. Sci. Monit. 15: CR511-CR517.
PMid:19789510
Saccucci P, Meloni GF, Verrotti A, Borgiani P, et al. (2010). A study of three polymorphic sites of the ADA gene in children with type 1 diabetes mellitus. J. Pediatr. Endocrinol. Metab. 23: 283-290.
http://dx.doi.org/10.1515/JPEM.2010.23.3.283
PMid:20480728
Safranow K, Rzeuski R, Binczak-Kuleta A, Czyzycka E, et al. (2007). ADA*2 allele of the adenosine deaminase gene may protect against coronary artery disease. Cardiology 108: 275-281.
http://dx.doi.org/10.1159/000099096
PMid:17287605
Sampaio-Barros PD, Azevedo VF, Bonfiglioli R, Campos WR, et al. (2007). Consenso Brasileiro de Espondiloartropatias: Espondilite Anquilosante e Artrite Psoriásica Diagnóstico e Tratamento - Primeira Revisão. Rev. Bras. Reumatol. 47: 233-242.
http://dx.doi.org/10.1590/S0482-50042007000400001
Sari RA, Taysi S, Yilmaz O and Bakan N (2003). Correlation of serum levels of adenosine deaminase activity and its isoenzymes with disease activity in rheumatoid arthritis. Clin. Exp. Rheumatol. 21: 87-90.
PMid:12673895
Sasaki S (1984). Pathogenetic significance of adenosine deaminase in autoimmune diseases. Nihon Seikeigeka Gakkai Zasshi 58: 219-230.
PMid:6332160
SEADE (2009). Fundação Sistema Estadual de Análise de Dados. Secretaria de Planejamento e Desenvolvimento Regional. Perfil Regional. Região Administrativa de São José do Rio Preto. Available at [http://www.seade.gov.br/produtos/perfil_regional/index.php]. Accessed March 30, 2011.
Sebastiani GD, Bottini N, Greco E, Saccucci P, et al. (2010). A study of Adenosine-Deaminase genetic polymorphism in rheumatoid arthritis. Int. J. Immunopathol. Pharmacol. 23: 791-795.
PMid:20943049
Van der Linden S and Van der Heijde D (1998). Ankylosing spondylitis: clinical features. Rheum. Dis. Clin. North Am. 24: 663-676.
http://dx.doi.org/10.1016/S0889-857X(05)70036-3
Wessels JA, Kooloos WM, De Jonge R, De Vries-Bouwstra JK, et al. (2006). Relationship between genetic variants in the adenosine pathway and outcome of methotrexate treatment in patients with recent-onset rheumatoid arthritis. Arthritis Rheum. 54: 2830-2839.
http://dx.doi.org/10.1002/art.22032
PMid:16947783