Publications

Found 1 results
Filters: Author is F.J.O. Paludo  [Clear All Filters]
2013
L. R. Fraga, Paludo, F. J. O., Bós, A. J. G., Ferraro, J. L. S., Dias, F. S., and Alho, C. S., More severe clinical course of cardiovascular dysfunction in intensive care unit patients with the 894TT eNOS genotype, vol. 12, pp. 562-568, 2013.
Antoniades C, Tousoulis D, Vasiliadou C, Pitsavos C, et al. (2005). Genetic polymorphism on endothelial nitric oxide synthase affects endothelial activation and inflammatory response during the acute phase of myocardial infarction. J. Am. Coll. Cardiol. 46: 1101-1109. http://dx.doi.org/10.1016/j.jacc.2005.05.072 PMid:16168297   Dafni C, Drakoulis N, Landt O, Panidis D, et al. (2010). Association of the eNOS E298D polymorphism and the risk of myocardial infarction in the Greek population. BMC Med. Genet. 11: 133. http://dx.doi.org/10.1186/1471-2350-11-133 PMid:20854685 PMCid:2954842   Fallavena PRV, Borges TJ, Paskulin DD, Thurow HS, et al. (2013). The synergy of -260T T CD14 and -308GG TNF-α genotypes in survival of critically ill patients. Scandinav. J. Immunol. 77: 62-68. http://dx.doi.org/10.1111/sji.12002 PMid:23036097   Ferreira FL, Bota DP, Bross A, Melot C, et al. (2001). Serial evaluation of the SOFA score to predict outcome in critically ill patients. JAMA 286: 1754-1758. http://dx.doi.org/10.1001/jama.286.14.1754 PMid:11594901   Hibi K, Ishigami T, Tamura K, Mizushima S, et al. (1998). Endothelial nitric oxide synthase gene polymorphism and acute myocardial infarction. Hypertension 32: 521-526. http://dx.doi.org/10.1161/01.HYP.32.3.521 PMid:9740620   Hingorani AD, Liang CF, Fatibene J, Lyon A, et al. (1999). A common variant of the endothelial nitric oxide synthase (Glu298→Asp) is a major risk factor for coronary artery disease in the UK. Circulation 100: 1515-1520. http://dx.doi.org/10.1161/01.CIR.100.14.1515 PMid:10510054   Huttunen R, Hurme M, Laine J, Eklund C, et al. (2009). Endothelial nitric oxide synthase G894T (GLU298ASP) polymorphism is associated with hypotension in patients with E. coli bacteremia but not in bacteremia caused by a gram-positive organism. Shock 31: 448-453. http://dx.doi.org/10.1097/SHK.0b013e318188e58e PMid:18827745   Iturry-Yamamoto GR, Moriguchi EH, Zago AC, Alho C, et al. (2007). Association of the 894G>T polymorphism of the endothelial constitutive nitric oxide synthase gene with unstable angina. Braz. J. Med. Biol. Res. 40: 475-483. http://dx.doi.org/10.1590/S0100-879X2006005000072 PMid:17401490   Kerkeni M, Addad F, Chauffert M, Myara A, et al. (2006). Hyperhomocysteinemia, endothelial nitric oxide synthase polymorphism, and risk of coronary artery disease. Clin. Chem. 52: 53-58. http://dx.doi.org/10.1373/clinchem.2005.057950 PMid:16284093   Knaus WA, Draper EA, Wagner DP and Zimmerman JE (1985). APACHE II: a severity of disease classification system. Crit. Care Med. 13: 818-829. http://dx.doi.org/10.1097/00003246-198510000-00009 PMid:3928249   Krishnagopalan S, Kumar A, Parrillo JE and Kumar A (2002). Myocardial dysfunction in the patient with sepsis. Curr. Opin. Crit. Care 8: 376-388. http://dx.doi.org/10.1097/00075198-200210000-00003 PMid:12357104   Lahiri DK and Nurnberger JI Jr (1991). A rapid non-enzymatic method for the preparation of HMW DNA from blood for RFLP studies. Nucleic Acids Res. 19: 5444. http://dx.doi.org/10.1093/nar/19.19.5444 PMid:1681511 PMCid:328920   Lembo G, De Luca N, Battagli C, Iovino G, et al. (2001). A common variant of endothelial nitric oxide synthase (Glu298Asp) is an independent risk factor for carotid atherosclerosis. Stroke 32: 735-740. http://dx.doi.org/10.1161/01.STR.32.3.735 PMid:11239195   Levy MM, Fink MP, Marshall JC, Abraham E, et al. (2003). 2001 SCCM/ESICM/ACCP/ATS/SIS International sepsis definitions conference. Intensive Care Med. 29: 530-538. PMid:12664219   Miyamoto Y, Saito Y, Kajiyama N, Yoshimura M, et al. (1998). Endothelial nitric oxide synthase gene is positively associated with essential hypertension. Hypertension 32: 3-8. http://dx.doi.org/10.1161/01.HYP.32.1.3 PMid:9674630   Moncada S and Higgs A (1993). The L-arginine-nitric oxide pathway. N. Engl. J. Med. 329: 2002-2012. http://dx.doi.org/10.1056/NEJM199312303292706 PMid:7504210   Nadaud S, Bonnardeaux A, Lathrop M and Soubrier F (1994). Gene structure, polymorphism and mapping of the human endothelial nitric oxide synthase gene. Biochem. Biophys. Res. Commun. 198: 1027-1033. http://dx.doi.org/10.1006/bbrc.1994.1146 PMid:7509596   Paludo FJO, Picanço JB, Fallavena PRV, Fraga LR, et al. (2013). Higher frequency of septic shock in septic patients with the 47C allele (rs4880) of the SOD2 gene. Gene 517: 106-111. http://dx.doi.org/10.1016/j.gene.2012.10.078 PMid:23142385   Parra FC, Amado RC, Lambertucci JR, Rocha J, et al. (2003). Color and genomic ancestry in Brazilians. Proc. Natl. Acad. Sci. U. S. A. 100: 177-182. http://dx.doi.org/10.1073/pnas.0126614100 PMid:12509516 PMCid:140919   Paskulin DD, Fallavena PRV, Paludo FJO, Borges TJ, et al. (2011). TNF -308G > a promoter polymorphism (rs1800629) and outcome from critical illness. Braz. J. Infect. Dis. 15: 231-238. http://dx.doi.org/10.1016/S1413-8670(11)70181-7   Tesauro M, Thompson WC, Rogliani P, Qi L, et al. (2000). Intracellular processing of endothelial nitric oxide synthase isoforms associated with differences in severity of cardiopulmonary diseases: cleavage of proteins with aspartate vs. glutamate at position 298. Proc. Natl. Acad. Sci. U. S. A. 97: 2832-2835. http://dx.doi.org/10.1073/pnas.97.6.2832 PMid:10717002 PMCid:16015   Umans JG and Levi R (1995). Nitric oxide in the regulation of blood flow and arterial pressure. Annu. Rev. Physiol. 57: 771-790. http://dx.doi.org/10.1146/annurev.ph.57.030195.004011 PMid:7539996   Veldman BA, Spiering W, Doevendans PA, Vervoort G, et al. (2002). The Glu298Asp polymorphism of the NOS 3 gene as a determinant of the baseline production of nitric oxide. J. Hypertens. 20: 2023-2027. http://dx.doi.org/10.1097/00004872-200210000-00022 PMid:12359981   Vincent JL, de Mendonça A, Cantraine F, Moreno R, et al. (1998). Use of the SOFA score to assess the incidence of organ dysfunction/failure in intensive care units: results of a multicenter, prospective study. Working group on "sepsis-related problems" of the European Society of Intensive Care Medicine. Crit. Care Med. 26: 1793-1800. http://dx.doi.org/10.1097/00003246-199811000-00016 PMid:9824069