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“Cathecol-O-methyl transferase Val158Met genotype is not a risk factor for conversion disorder”, vol. 12, pp. 852-858, 2013.
, Albaugh MD, Harder VS, Althoff RR, Rettew DC, et al. (2010). COMT Val158Met genotype as a risk factor for problem behaviors in youth. J. Am. Acad. Child Adolesc. Psychiatry 49: 841-849.
http://dx.doi.org/10.1016/j.jaac.2010.05.015
PMid:20643317 PMCid:3141335
American Psychiatric Association (2000). Diagnostic and Statistical Manual of Mental Disorders, DSM-IV-TP. 4th edn. Text Revision. American Psychiatric Association, Washington.
Binzer M, Andersen PM and Kullgren G (1997). Clinical characteristics of patients with motor disability due to conversion disorder: a prospective control group study. J. Neurol. Neurosurg. Psychiatry 63: 83-88.
http://dx.doi.org/10.1136/jnnp.63.1.83
PMid:9221972 PMCid:2169635
Caspi A, Langley K, Milne B, Moffitt TE, et al. (2008). A replicated molecular genetic basis for subtyping antisocial behavior in children with attention-deficit/hyperactivity disorder. Arch. Gen. Psychiatry 65: 203-210.
http://dx.doi.org/10.1001/archgenpsychiatry.2007.24
PMid:18250258
Chen J, Lipska BK, Halim N, Ma QD, et al. (2004). Functional analysis of genetic variation in catechol-O-methyltransferase (COMT): effects on mRNA, protein, and enzyme activity in postmortem human brain. Am. J. Hum. Genet. 75: 807- 821.
http://dx.doi.org/10.1086/425589
PMid:15457404 PMCid:1182110
Craddock N, Owen MJ and O'Donovan MC (2006). The catechol-O-methyl transferase (COMT) gene as a candidate for psychiatric phenotypes: evidence and lessons. Mol. Psychiatry 11: 446-458.
http://dx.doi.org/10.1038/sj.mp.4001808
PMid:16505837
DeYoung CG, Getchell M, Koposov RA, Yrigollen CM, et al. (2010). Variation in the catechol-O-methyltransferase Val 158 Met polymorphism associated with conduct disorder and ADHD symptoms, among adolescent male delinquents. Psychiatr. Genet. 20: 20-24.
http://dx.doi.org/10.1097/YPG.0b013e32833511e4
PMid:19997043 PMCid:2895253
Dula DJ and DeNaples L (1995). Emergency department presentation of patients with conversion disorder. Acad. Emerg. Med. 2: 120-123.
http://dx.doi.org/10.1111/j.1553-2712.1995.tb03174.x
PMid:7621217
Eisenberg J, Mei-Tal G, Steinberg A, Tartakovsky E, et al. (1999). Haplotype relative risk study of catechol-O-methyltransferase (COMT) and attention deficit hyperactivity disorder (ADHD): association of the high-enzyme activity Val allele with ADHD impulsive-hyperactive phenotype. Am. J. Med. Genet. 88: 497-502.
http://dx.doi.org/10.1002/(SICI)1096-8628(19991015)88:5<497::AID-AJMG12>3.0.CO;2-F
Glatt SJ, Faraone SV and Tsuang MT (2003). Association between a functional catechol O-methyltransferase gene polymorphism and schizophrenia: meta-analysis of case-control and family-based studies. Am. J. Psychiatry 160: 469-476.
http://dx.doi.org/10.1176/appi.ajp.160.3.469
PMid:12611827
Gogos JA, Morgan M, Luine V, Santha M, et al. (1998). Catechol-O-methyltransferase-deficient mice exhibit sexually dimorphic changes in catecholamine levels and behavior. Proc. Natl. Acad. Sci. U. S. A. 95: 9991-9996.
http://dx.doi.org/10.1073/pnas.95.17.9991
PMid:9707588 PMCid:21449
Gutierrez B, Bertranpetit J, Guillamat R, Valles V, et al. (1997). Association analysis of the catechol O-methyltransferase gene and bipolar affective disorder. Am. J. Psychiatry 154: 113-115.
PMid:8988970
Holmboe K, Nemoda Z, Fearon RM, Csibra G, et al. (2010). Polymorphisms in dopamine system genes are associated with individual differences in attention in infancy. Dev. Psychol. 46: 404-416.
http://dx.doi.org/10.1037/a0018180
PMid:20210499 PMCid:3276838
Kereszturi E, Tarnok Z, Bognar E, Lakatos K, et al. (2008). Catechol-O-methyltransferase Val158Met polymorphism is associated with methylphenidate response in ADHD children. Am. J. Med. Genet. B Neuropsychiatr. Genet. 147B: 1431-1435.
http://dx.doi.org/10.1002/ajmg.b.30704
PMid:18214865
Kunugi H, Vallada HP, Hoda F, Kirov G, et al. (1997). No evidence for an association of affective disorders with high- or low-activity allele of catechol-o-methyltransferase gene. Biol. Psychiatry 42: 282-285.
http://dx.doi.org/10.1016/S0006-3223(96)00366-6
Lachman HM, Papolos DF, Saito T, Yu YM, et al. (1996). Human catechol-O-methyltransferase pharmacogenetics: description of a functional polymorphism and its potential application to neuropsychiatric disorders. Pharmacogenetics 6: 243-250.
http://dx.doi.org/10.1097/00008571-199606000-00007
PMid:8807664
Lewis DA, Melchitzky DS, Sesack SR, Whitehead RE, et al. (2001). Dopamine transporter immunoreactivity in monkey cerebral cortex: regional, laminar, and ultrastructural localization. J. Comp. Neurol. 432: 119-136.
http://dx.doi.org/10.1002/cne.1092
PMid:11241381
Lotta T, Vidgren J, Tilgmann C, Ulmanen I, et al. (1995). Kinetics of human soluble and membrane-bound catechol O-methyltransferase: a revised mechanism and description of the thermolabile variant of the enzyme. Biochemistry 34: 4202-4210.
http://dx.doi.org/10.1021/bi00013a008
PMid:7703232
Monuteaux MC, Biederman J, Doyle AE, Mick E, et al. (2009). Genetic risk for conduct disorder symptom subtypes in an ADHD sample: specificity to aggressive symptoms. J. Am. Acad. Child Adolesc. Psychiatry 48: 757-764.
http://dx.doi.org/10.1097/CHI.0b013e3181a5661b
PMid:19465875
Palmason H, Moser D, Sigmund J, Vogler C, et al. (2010). Attention-deficit/hyperactivity disorder phenotype is influenced by a functional catechol-O-methyltransferase variant. J. Neural Transm. 117: 259-267.
http://dx.doi.org/10.1007/s00702-009-0338-2
PMid:19946713
Purcell TB (1991). The somatic patient. Emerg. Med. Clin. North Am. 9: 137-159.
PMid:2001663
Sadock BJ and Sadock VA (2000). Kaplan and Sadock's Comprehensive Textbook of Psychiatry. 7th edn. Lippincott Williams & Wilkins, Baltimore.
Sengupta S, Grizenko N, Schmitz N, Schwartz G, et al. (2008). COMT Val108/158Met polymorphism and the modulation of task-oriented behavior in children with ADHD. Neuropsychopharmacology 33: 3069-3077.
http://dx.doi.org/10.1038/npp.2008.85
PMid:18580877 PMCid:2885152
Shifman S, Bronstein M, Sternfeld M, Pisante A, et al. (2004). COMT: a common susceptibility gene in bipolar disorder and schizophrenia. Am. J. Med. Genet. B Neuropsychiatr. Genet. 128B: 61-64.
http://dx.doi.org/10.1002/ajmg.b.30032
PMid:15211633
Stonnington CM, Barry JJ and Fisher RS (2006). Conversion disorder. Am. J. Psychiatry 163: 1510-1517.
http://dx.doi.org/10.1176/appi.ajp.163.9.1510
PMid:16946174
Tunbridge EM, Bannerman DM, Sharp T and Harrison PJ (2004). Catechol-o-methyltransferase inhibition improves set-shifting performance and elevates stimulated dopamine release in the rat prefrontal cortex. J. Neurosci. 24: 5331- 5335.
http://dx.doi.org/10.1523/JNEUROSCI.1124-04.2004
PMid:15190105
Weinshilboum RM, Otterness DM and Szumlanski CL (1999). Methylation pharmacogenetics: catechol O-methyltransferase, thiopurine methyltransferase, and histamine N-methyltransferase. Annu. Rev. Pharmacol. Toxicol. 39: 19-52.
http://dx.doi.org/10.1146/annurev.pharmtox.39.1.19
PMid:10331075
Wirgenes KV, Djurovic S, Sundet K, Agartz I, et al. (2010). Catechol O-methyltransferase variants and cognitive performance in schizophrenia and bipolar disorder versus controls. Schizophr. Res. 122: 31-37.
http://dx.doi.org/10.1016/j.schres.2010.05.007
PMid:20605701