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C. L. Li, Du, X. Y., Gao, J., Wang, C., Guo, H. G., Dai, F. W., Sa, X. Y., An, W., Chen, Z. W., Li, C. L., Du, X. Y., Gao, J., Wang, C., Guo, H. G., Dai, F. W., Sa, X. Y., An, W., and Chen, Z. W., Phylogenetic analysis of the Mongolian gerbil (Meriones unguiculatus) from China based on mitochondrial genome, vol. 15, p. -, 2016.
C. L. Li, Du, X. Y., Gao, J., Wang, C., Guo, H. G., Dai, F. W., Sa, X. Y., An, W., Chen, Z. W., Li, C. L., Du, X. Y., Gao, J., Wang, C., Guo, H. G., Dai, F. W., Sa, X. Y., An, W., and Chen, Z. W., Phylogenetic analysis of the Mongolian gerbil (Meriones unguiculatus) from China based on mitochondrial genome, vol. 15, p. -, 2016.
L. Li, Li, Y., Zeng, X. C., Li, J., and Du, X. Y., Role of interleukin-4 genetic polymorphisms and environmental factors in the risk of asthma in children, vol. 15, no. 4, p. -, 2016.
Conflicts of interestThe authors declare no conflict of interest.REFERENCESAkinbami LJ, Schoendorf KC, et al (2002). Trends in childhood asthma: prevalence, health care utilization, and mortality. Pediatrics 110: 315-322. Al-Mazam A, Mohamed AG, et al (2001). Risk factors of bronchial asthma in bahrah, saudi arabia. J. Family Community Med. 8: 33-39. Andrews RP, Burrell L, Rosa-Rosa L, Cunningham CM, et al (2001). Analysis of the Ser786Pro interleukin-4 receptor alpha allelic variant in allergic and nonallergic asthma and its functional consequences. Clin. Immunol. 100: 298-304. Antczak A, Domańska-Senderowska D, Górski P, Pastuszak-Lewandoska D, et al (2016). Analysis of changes in expression of IL-4/IL-13/STAT6 pathway and correlation with the selected clinical parameters in patients with atopic asthma. Int. J. Immunopathol. Pharmacol. 29: 195-204. Bal SM, Bernink JH, Nagasawa M, Groot J, et al (2016). IL-1β, IL-4 and IL-12 control the fate of group 2 innate lymphoid cells in human airway inflammation in the lungs. Nat. Immunol. 17: 636-645. Barner M, Mohrs M, Brombacher F, Kopf M, et al (1998). Differences between IL-4R alpha-deficient and IL-4-deficient mice reveal a role for IL-13 in the regulation of Th2 responses. Curr. Biol. 8: 669-672. Borish L, et al (2010). IL-4 and IL-13 dual antagonism: a promising approach to the dilemma of generating effective asthma biotherapeutics. Am. J. Respir. Crit. Care Med. 181: 769-770. Boulet LP, FitzGerald JM, Levy ML, Cruz AA, et al (2012). A guide to the translation of the Global Initiative for Asthma (GINA) strategy into improved care. Eur. Respir. J. 39: 1220-1229. Chen ZH, Wang PL, Shen HH, et al (2013). Asthma research in China: a five-year review. Respirology 18 (Suppl 3): 10-19. Cook J, Saglani S, et al (2016). Pathogenesis and prevention strategies of severe asthma exacerbations in children. Curr. Opin. Pulm. Med. 22: 25-31. Cui T, Wu J, Pan S, Xie J, et al (2003). Polymorphisms in the IL-4 and IL-4R [alpha] genes and allergic asthma. Clin. Chem. Lab. Med. 41: 888-892. Demirdag YY, Ramadan HH, et al (2016). Direct measurement of upper airway inflammation in children with chronic rhinosinusitis: implications for asthma. Curr. Opin. Allergy Clin. Immunol. 16: 18-23. Eaton DK, Kann L, Kinchen S, Shanklin S, Centers for Disease Control and Prevention (CDC)et al (2008). Youth risk behavior surveillance--United States, 2007. MMWR Surveill. Summ. 57: 1-131. Gonzalez-Garcia M, Caballero A, Jaramillo C, Maldonado D, et al (2015). Prevalence, risk factors and underdiagnosis of asthma and wheezing in adults 40 years and older: A population-based study. J. Asthma 52: 823-830. Hosseini-Farahabadi S, Tavakkol-Afshari J, Rafatpanah H, Farid Hosseini R, et al (2007). Association between the polymorphisms of IL-4 gene promoter (-590C>T), IL-13 coding region (R130Q) and IL-16 gene promoter (-295T>C) and allergic asthma. Iran. J. Allergy Asthma Immunol. 6: 9-14. Hovland V, Riiser A, Mowinckel P, Carlsen KH, et al (2015). Early risk factors for pubertal asthma. Clin. Exp. Allergy 45: 164-176. Huang ZY, Cheng BJ, Cai GJ, Zhang BF, et al (2015). Association of the IL-4R Q576R polymorphism and asthma in the Chinese Han population: a meta-analysis. Genet. Mol. Res. 14: 2900-2911. Jaakkola MS, Jaakkola JJ, et al (2012). Assessment of public health impact of work-related asthma. BMC Med. Res. Methodol. 12: 22. Kamali-Sarvestani E, Ghayomi MA, Nekoee A, et al (2007). Association of TNF-alpha -308 G/A and IL-4 -589 C/T gene promoter polymorphisms with asthma susceptibility in the south of Iran. J. Investig. Allergol. Clin. Immunol. 17: 361-366. Lee JH, Park HS, Park SW, Jang AS, et al (2004). ADAM33 polymorphism: association with bronchial hyper-responsiveness in Korean asthmatics. Clin. Exp. Allergy 34: 860-865. Li Y, Guo B, Zhang L, Han J, et al (2008). Association between C-589T polymorphisms of interleukin-4 gene promoter and asthma: a meta-analysis. Respir. Med. 102: 984-992. Mak JC, Ko FW, Chu CM, Leung HC, et al (2007). Polymorphisms in the IL-4, IL-4 receptor alpha chain, TNF-alpha, and lymphotoxin-alpha genes and risk of asthma in Hong Kong Chinese adults. Int. Arch. Allergy Immunol. 144: 114-122. Masoli M, Fabian D, Holt S, Beasley R, Global Initiative for Asthma (GINA) Programet al (2004). The global burden of asthma: executive summary of the GINA Dissemination Committee report. Allergy 59: 469-478. Micheal S, Minhas K, Ishaque M, Ahmed F, et al (2013). IL-4 gene polymorphisms and their association with atopic asthma and allergic rhinitis in Pakistani patients. J. Investig. Allergol. Clin. Immunol. 23: 107-111. Nambu M, Holgate S, et al (2009). Differences in the asthma treatment of children between europe and Japan: a questionnaire-based survey using model cases. World Allergy Organ. J. 2: 54-56. Osei-Kumah A, Ammit AJ, Smith R, Ge Q, et al (2006). Inflammatory mediator release in normal bronchial smooth muscle cells is altered by pregnant maternal and fetal plasma independent of asthma. Placenta 27: 847-852. Pattnaik B, Bodas M, Bhatraju NK, Ahmad T, et al (2016). IL-4 promotes asymmetric dimethylarginine accumulation, oxo-nitrative stress, and hypoxic response-induced mitochondrial loss in airway epithelial cells. J. Allergy Clin. Immunol. 138: 130-141.e9. Rosa-Rosa L, Zimmermann N, Bernstein JA, Rothenberg ME, et al (1999). The R576 IL-4 receptor alpha allele correlates with asthma severity. J. Allergy Clin. Immunol. 104: 1008-1014. Sengler C, Lau S, Wahn U, Nickel R, et al (2002). Interactions between genes and environmental factors in asthma and atopy: new developments. Respir. Res. 3: 7. Smallwood DT, Apollonio B, Willimott S, Lezina L, et al (2016). Extracellular vesicles released by CD40/IL-4-stimulated CLL cells confer altered functional properties to CD4+ T cells. Blood 128: 542-552. Tang L, Lin HG, Chen BF, et al (2014). Association of IL-4 promoter polymorphisms with asthma: a meta-analysis. Genet. Mol. Res. 13: 1383-1394. Toskala E, Kennedy DW, et al (2015). Asthma risk factors. Int. Forum Allergy Rhinol. 5 (Suppl 1): S11-S16. Tulah AS, Holloway JW, Sayers I, et al (2013). Defining the contribution of SNPs identified in asthma GWAS to clinical variables in asthmatic children. BMC Med. Genet. 14: 100. Wills-Karp M, Luyimbazi J, Xu X, Schofield B, et al (1998). Interleukin-13: central mediator of allergic asthma. Science 282: 2258-2261. Wongratanacheewin S, et al (2014). Update of cytokines and genes in asthma and allergic rhinitis. Asian Pac. J. Allergy Immunol. 32: 275. Zahran HS, Bailey C, et al (2013). Factors associated with asthma prevalence among racial and ethnic groups--United States, 2009-2010 behavioral risk factor surveillance system. J. Asthma 50: 583-589.