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C. - P. Liu, Jiang, J. - A., Wang, T., Liu, X. - M., Gao, L., Zhu, R. - R., Shen, Y., Wu, M., Xu, T., and Zhang, X. - G., CTLA-4 and CD86 genetic variants and haplotypes in patients with rheumatoid arthritis in southeastern China, vol. 12, pp. 1373-1382, 2013.
Abdallah AM, Renzoni EA, Anevlavis S, Lagan AL, et al. (2006). A polymorphism in the promoter region of the CD86 (B7.2) gene is associated with systemic sclerosis. Int. J. Immunogenet. 33: 155-161. PMid:16712644   Almasi S, Erfani N, Mojtahedi Z, Rajaee A, et al. (2006). Association of CTLA-4 gene promoter polymorphisms with systemic sclerosis in Iranian population. Genes Immun. 7: 401-406. PMid:16775619   Arnett FC, Edworthy SM, Bloch DA, McShane DJ, et al. (1988). The American Rheumatism Association 1987 revised criteria for the classification of rheumatoid arthritis. Arthritis Rheum. 31: 315-324. PMid:3358796   Catalan D, Aravena O, Sabugo F, Wurmann P, et al. (2010). B cells from rheumatoid arthritis patients show important alterations in the expression of CD86 and FcgammaRIIb, which are modulated by anti-tumor necrosis factor therapy. Arthritis Res. Ther. 12: R68. PMid:20398308 PMCid:2888223   Chang JC, Liu CA, Chuang H, Ou CY, et al. (2004). Gender-limited association of cytotoxic T-lymphocyte antigen-4 (CTLA-4) polymorphism with cord blood IgE levels. Pediatr. Allergy Immunol. 15: 506-512. PMid:15610363   Fox D (2005). Etiology and Pathogenesis of Rheumatoid Arthritis. In: Arthritis and Allied Conditions (Koopman W, ed.). Lippincott Williams & Wilkins, Philadephia, 1089-1115.   Haimila K, Einarsdottir E, de Kauwe A, Koskinen LL, et al. (2009). The shared CTLA4-ICOS risk locus in celiac disease, IgA deficiency and common variable immunodeficiency. Genes Immun. 10: 151-161. PMid:19020530   Howson JM, Walker NM, Smyth DJ and Todd JA (2009). Analysis of 19 genes for association with type I diabetes in the Type I Diabetes Genetics Consortium families. Genes Immun. 10 (Suppl 1): S74-S84. PMid:19956106 PMCid:2810493   Jones AL, Holliday EG, Mowry BJ, McLean DE, et al. (2009). CTLA-4 single-nucleotide polymorphisms in a Caucasian population with schizophrenia. Brain Behav. Immun. 23: 347-350. PMid:18848621   Kouki T, Gardine CA, Yanagawa T and Degroot LJ (2002). Relation of three polymorphisms of the CTLA-4 gene in patients with Graves' disease. J. Endocrinol. Invest. 25: 208-213. PMid:11936461   Kusztal M, Kościelska-Kasprzak K, Drulis-Fajdasz D, Magott-Procelewska M, et al. (2010). The influence of CTLA-4 gene polymorphism on long-term kidney allograft function in Caucasian recipients. Transpl. Immunol. 23: 121-124. PMid:20470888   Landi D, Moreno V, Guino E, Vodicka P, et al. (2011). Polymorphisms affecting micro-RNA regulation and associated with the risk of dietary-related cancers: a review from the literature and new evidence for a functional role of rs17281995 (CD86) and rs1051690 (INSR), previously associated with colorectal cancer. Mutat. Res. 717: 109-115. PMid:20971123   Liang YL, Wu H, Li PQ, Xie XD, et al. (2011). Signal transducer and activator of transcription 4 gene polymorphisms associated with rheumatoid arthritis in Northwestern Chinese Han population. Life Sci. 89: 171-175. PMid:21683716   Ligers A, Teleshova N, Masterman T, Huang WX, et al. (2001). CTLA-4 gene expression is influenced by promoter and exon 1 polymorphisms. Genes Immun. 2: 145-152. PMid:11426323   Liu MF, Kohsaka H, Sakurai H, Azuma M, et al. (1996). The presence of costimulatory molecules CD86 and CD28 in rheumatoid arthritis synovium. Arthritis Rheum. 39: 110-114. PMid:8546719   Liu Y, Liang WB, Gao LB, Pan XM, et al. (2010). CTLA4 and CD86 gene polymorphisms and susceptibility to chronic obstructive pulmonary disease. Hum. Immunol. 71: 1141-1146. PMid:20732370   Magistrelli G, Jeannin P, Herbault N, Benoit De CA, et al. (1999). A soluble form of CTLA-4 generated by alternative splicing is expressed by nonstimulated human T cells. Eur. J. Immunol. 29: 3596-3602.<3596::AID-IMMU3596>3.0.CO;2-Y   Marin LA, Moya-Quiles MR, Miras M, Muro M, et al. (2005). Evaluation of CD86 gene polymorphism at +1057 position in liver transplant recipients. Transpl. Immunol. 15: 69-74. PMid:16223675   Matsushita M, Tsuchiya N, Oka T, Yamane A, et al. (2000). New polymorphisms of human CD80 and CD86: lack of association with rheumatoid arthritis and systemic lupus erythematosus. Genes Immun. 1: 428-434. PMid:11196673   Maurer M, Loserth S, Kolb-Maurer A, Ponath A, et al. (2002). A polymorphism in the human cytotoxic T-lymphocyte antigen 4 (CTLA4) gene (exon 1 +49) alters T-cell activation. Immunogenetics 54: 1-8. PMid:11976786   Orozco G, Rueda B and Martin J (2006). Genetic basis of rheumatoid arthritis. Biomed. Pharmacother. 60: 656-662. PMid:17055211   Pawlak E, Karabon L, Wlodarska-Polinska I, Jedynak A, et al. (2010). Influence of CTLA-4/CD28/ICOS gene polymorphisms on the susceptibility to cervical squamous cell carcinoma and stage of differentiation in the Polish population. Hum. Immunol. 71: 195-200. PMid:19913589   Plant D, Flynn E, Mbarek H, Dieude P, et al. (2010). Investigation of potential non-HLA rheumatoid arthritis susceptibility loci in a European cohort increases the evidence for nine markers. Ann. Rheum. Dis. 69: 1548-1553. PMid:20498205 PMCid:2938898   Rai E and Wakeland EK (2011). Genetic predisposition to autoimmunity - what have we learned? Semin. Immunol. 23: 67-83. PMid:21288738   Scalapino KJ and Daikh DI (2008). CTLA-4: a key regulatory point in the control of autoimmune disease. Immunol. Rev. 223: 143-155. PMid:18613834   Sharpe AH and Freeman GJ (2002). The B7-CD28 superfamily. Nat. Rev. Immunol. 2: 116-126. PMid:11910893   Sole X, Guino E, Valls J, Iniesta R, et al. (2006). SNPStats: a web tool for the analysis of association studies. Bioinformatics 22: 1928-1929. PMid:16720584   Su TH, Chang TY, Lee YJ, Chen CK, et al. (2007). CTLA-4 gene and susceptibility to human papillomavirus-16- associated cervical squamous cell carcinoma in Taiwanese women. Carcinogenesis 28: 1237-1240. PMid:17341658   Tivol EA, Borriello F, Schweitzer AN, Lynch WP, et al. (1995). Loss of CTLA-4 leads to massive lymphoproliferation and fatal multiorgan tissue destruction, revealing a critical negative regulatory role of CTLA-4. Immunity 3: 541-547.   Wang XB, Zhao X, Giscombe R and Lefvert AK (2002). A CTLA-4 gene polymorphism at position -318 in the promoter region affects the expression of protein. Genes Immun. 3: 233-234. PMid:12058260   Yadav D and Sarvetnick N (2007). B7-2 regulates survival, phenotype, and function of APCs. J. Immunol. 178: 6236- 6241. PMid:17475851   Zaletel K, Krhin B, Gaberscek S and Hojker S (2006). Thyroid autoantibody production is influenced by exon 1 and promoter CTLA-4 polymorphisms in patients with Hashimoto's thyroiditis. Int. J. Immunogenet. 33: 87-91. PMid:16611252
Y. L. Hou, Ding, X., Hou, W., Song, B., Wang, T., Wang, F., Li, J., Zhong, J., Xu, T., Ma, B. X., Zhu, H. Q., Li, J. H., and Zhong, J. C., Overexpression, purification, and pharmacologic evaluation of anticancer activity of ribosomal protein L24 from the giant panda (Ailuropoda melanoleuca), vol. 12, pp. 4735-4750, 2013.