Publications
Found 6 results
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“Gender and ethnicity modify the association between the CYP1A2 rs762551 polymorphism and habitual coffee intake: evidence from a meta-analysis”, vol. 15, p. -, 2016.
, , , “Mn-SOD 47 CC genotype in combination with high tea consumption may prevent complications in Tunisian type-2 diabetes”, vol. 14, pp. 8613-8622, 2015.
, “Bias toward a 1:1 ratio in primer-introduced restriction analysis PCR: mechanism and minimization”, vol. 13, pp. 5686-5694, 2014.
, “Relationship between glutathione S-transferase P1 polymorphisms and chronic obstructive pulmonary disease in a Tunisian population”, vol. 9, pp. 897-907, 2010.
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Calikoglu M, Tamer L, Ates AN, Karakas S, et al. (2006). The association between polymorphic genotypes of glutathione S-transferases and COPD in the Turkish population. Biochem. Genet. 44: 307-319.
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Chan-Yeung M, Ho SP, Cheung AH, So LK, et al. (2007). Polymorphisms of glutathione S-transferase genes and functional activity in smokers with or without COPD. Int. J. Tuberc. Lung Dis. 11: 508-514.
PMid:17439673
Cheng SL, Yu CJ, Chen CJ and Yang PC (2004). Genetic polymorphism of epoxide hydrolase and glutathione S-transferase in COPD. Eur. Respir. J. 23: 818-824.
http://dx.doi.org/10.1183/09031936.04.00104904
PMid:15218992
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Eaton DL and Bammler TK (1999). Concise review of the glutathione S-transferases and their significance to toxicology. Toxicol. Sci. 49: 156-164.
http://dx.doi.org/10.1093/toxsci/49.2.156
PMid:10416260
Global Initiative for Chronic Obstructive Lung Disease (GOLD) (2006). Global Strategy for the Diagnosis, Mangement and Prevention of Chronic Obstructive Pulmonary Disease. Available at [http://www.goldcopd.com]. Accessed April 14, 2009.
Hanene C, Jihene L, Jamel A, Kamel H, et al. (2007). Association of GST genes polymorphisms with asthma in Tunisian children. Mediators Inflamm. 2007: 19564. [DOI: 10.1155/2007/19564].
http://dx.doi.org/10.1155/2007/19564
Harries LW, Stubbins MJ, Forman D, Howard GC, et al. (1997). Identification of genetic polymorphisms at the glutathione S-transferase Pi locus and association with susceptibility to bladder, testicular and prostate cancer. Carcinogenesis 18: 641-644.
http://dx.doi.org/10.1093/carcin/18.4.641
PMid:9111193
He JQ, Connett JE, Anthonisen NR, Pare PD, et al. (2004). Glutathione S-transferase variants and their interaction with smoking on lung function. Am. J. Respir. Crit. Care Med. 170: 388-394.
http://dx.doi.org/10.1164/rccm.200312-1763OC
PMid:15184197
Hu X, O'Donnell R, Srivastava SK, Xia H, et al. (1997). Active site architecture of polymorphic forms of human glutathione S-transferase P1-1 accounts for their enantioselectivity and disparate activity in the glutathione conjugation of 7beta,8alpha-dihydroxy-9alpha,10alpha-oxy-7,8,9,10-tetrahydrobenzo(a)pyrene. Biochem. Biophys. Res. Commun. 235: 424-428.
http://dx.doi.org/10.1006/bbrc.1997.6777
PMid:9199210
Ishii T, Matsuse T, Teramoto S, Matsui H, et al. (1999). Glutathione S-transferase P1 (GSTP1) polymorphism in patients with chronic obstructive pulmonary disease. Thorax 54: 693-696.
http://dx.doi.org/10.1136/thx.54.8.693
PMid:10413721 PMCid:1745535
Johansson AS, Stenberg G, Widersten M and Mannervik B (1998). Structure-activity relationships and thermal stability of human glutathione transferase P1-1 governed by the H-site residue 105. J. Mol. Biol. 278: 687-698.
http://dx.doi.org/10.1006/jmbi.1998.1708
PMid:9600848
Katoh T, Kaneko S, Takasawa S, Nagata N, et al. (1999). Human glutathione S-transferase P1 polymorphism and susceptibility to smoking related epithelial cancer; oral, lung, gastric, colorectal and urothelial cancer. Pharmacogenetics 9: 165-169.
PMid:10376763
Lokke A, Lange P, Scharling H, Fabricius P, et al. (2006). Developing COPD: a 25 year follow-up study of the general population. Thorax 61: 935-939.
http://dx.doi.org/10.1136/thx.2006.062802
PMid:17071833 PMCid:2121175
Morita S, Yano M, Tsujinaka T, Ogawa A, et al. (1998). Association between genetic polymorphisms of glutathione S-transferase P1 and N-acetyltransferase 2 and susceptibility to squamous-cell carcinoma of the esophagus. Int. J. Cancer 79: 517-520.
http://dx.doi.org/10.1002/(SICI)1097-0215(19981023)79:5<517::AID-IJC12>3.0.CO;2-Z
Murray CJ and Lopez AD (1997). Alternative projections of mortality and disability by cause 1990-2020: Global Burden of Disease Study. Lancet 349: 1498-1504.
http://dx.doi.org/10.1016/S0140-6736(96)07492-2
Nebert DW and Vasiliou V (2004). Analysis of the glutathione S-transferase (GST) gene family. Hum. Genomics 1: 460-464.
http://dx.doi.org/10.1186/1479-7364-1-6-460
PMid:15607001 PMCid:3500200
Pauwels RA and Rabe KF (2004). Burden and clinical features of chronic obstructive pulmonary disease (COPD). Lancet 364: 613-620.
http://dx.doi.org/10.1016/S0140-6736(04)16855-4
Rodríguez F, de la Roza C, Jardi R, Schaper M, et al. (2005). Glutathione S-transferase P1 and lung function in patients with alpha1-antitrypsin deficiency and COPD. Chest 127: 1537-1543.
http://dx.doi.org/10.1378/chest.127.5.1537
PMid:15888825
Ryberg D, Skaug V, Hewer A, Phillips DH, et al. (1997). Genotypes of glutathione transferase M1 and P1 and their significance for lung DNA adduct levels and cancer risk. Carcinogenesis 18: 1285-1289.
http://dx.doi.org/10.1093/carcin/18.7.1285
PMid:9230269
Smolonska J, Wijmenga C, Postma DS and Boezen HM (2009). Meta-analyses on suspected chronic obstructive pulmonary disease genes: a summary of 20 years' research. Am. J. Respir. Crit. Care Med. 180: 618-631.
http://dx.doi.org/10.1164/rccm.200905-0722OC
PMid:19608716
Strange RC, Spiteri MA, Ramachandran S and Fryer AA (2001). Glutathione-S-transferase family of enzymes. Mutat. Res. 482: 21-26.
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Sundberg K, Johansson AS, Stenberg G, Widersten M, et al. (1998). Differences in the catalytic efficiencies of allelic variants of glutathione transferase P1-1 towards carcinogenic diol epoxides of polycyclic aromatic hydrocarbons. Carcinogenesis 19: 433-436.
http://dx.doi.org/10.1093/carcin/19.3.433
PMid:9525277
Tamer L, Calikoglu M, Ates NA, Yildirim H, et al. (2004). Glutathione-S-transferase gene polymorphisms (GSTT1, GSTM1, GSTP1) as increased risk factors for asthma. Respirology 9: 493-498.
http://dx.doi.org/10.1111/j.1440-1843.2004.00657.x
PMid:15612961
van Lieshout EM, Roelofs HM, Dekker S, Mulder CJ, et al. (1999). Polymorphic expression of the glutathione S-transferase P1 gene and its susceptibility to Barrett's esophagus and esophageal carcinoma. Cancer Res. 59: 586-589.
PMid:9973204
Watson MA, Stewart RK, Smith GB, Massey TE, et al. (1998). Human glutathione S-transferase P1 polymorphisms: relationship to lung tissue enzyme activity and population frequency distribution. Carcinogenesis 19: 275-280.
http://dx.doi.org/10.1093/carcin/19.2.275
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http://dx.doi.org/10.1055/s-2007-1007169
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Zimniak P, Nanduri B, Pikula S, Bandorowicz-Pikula J, et al. (1994). Naturally occurring human glutathione S-transferase GSTP1-1 isoforms with isoleucine and valine in position 104 differ in enzymic properties. Eur. J. Biochem. 224: 893-899.
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PMid:7925413