Publications

Found 6 results
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2015
H. R. Dong, Li, H. S., Wang, S. C., Balin, Q. M., and Chang, P. Y., “Association between PDCD1, CTLA4, and MECP2 gene polymorphisms and systemic lupus erythematosus in the Chinese Northern Han”, vol. 14, pp. 17567-17573, 2015.
P. Y. Chang, Liu, Z. Y., Qin, L., and Zhao, P., “Association of NPRA and NPRC gene variants and hypertension in Mongolian population”, vol. 14, pp. 18494-18502, 2015.
P. Y. Chang, Qin, L., Zhao, P., and Liu, Z. Y., “Association of regulator of G protein signaling (RGS5) gene variants and essential hypertension in Mongolian and Han populations”, vol. 14, pp. 17641-17650, 2015.
L. Yang, Tian, R. G., Chang, P. Y., Yan, M. R., and Su, X. L., “Association of SNPs in the PPARγ gene and hypertension in a Mongolian population”, vol. 14, pp. 19295-19308, 2015.
2011
P. Y. Chang, Zhao, L. G., and Su, X. L., “Association of TSC gene variants and hypertension in Mongolian and Han populations”, vol. 10, pp. 902-909, 2011.
Cardon LR and Abecasis GR (2003). Using haplotype blocks to map human complex trait loci. Trends Genet. 19: 135-140. doi:10.1016/S0168-9525(03)00022-2 Carlson CS, Eberle MA, Rieder MJ, Yi Q, et al. (2004). Selecting a maximally informative set of single-nucleotide polymorphisms for association analyses using linkage disequilibrium. Am. J. Hum. Genet. 74: 106-120. doi:10.1086/381000 PMid:14681826 Crosio C, Cecconi F, Mariottini P, Cesareni G, et al. (1996). Fugu intron oversize reveals the presence of U15 snoRNA coding sequences in some introns of the ribosomal protein S3 gene. Genome Res. 6: 1227-1231. doi:10.1101/gr.6.12.1227 PMid:8973918 Fava C, Montagnana M, Rosberg L, Burri P, et al. (2008). Subjects heterozygous for genetic loss of function of the thiazide-sensitive cotransporter have reduced blood pressure. Hum. Mol. Genet. 17: 413-418. doi:10.1093/hmg/ddm318 PMid:17981812 Jacob-Ferreira AL and Sandrim VC (2007). Endothelial nitric oxide synthase polymorphisms and hypertension: improved clinical evidence derived from haplotype analysis. Int. J. Cardiol. 116: 116. doi:10.1016/j.ijcard.2006.11.048 PMid:17223211 Keszei AP, Tisler A, Backx PH, Andrulis IL, et al. (2007). Molecular variants of the thiazide-sensitive Na+-Cl- cotransporter in hypertensive families. J. Hypertens. 25: 2074-2081. doi:10.1097/HJH.0b013e3282a9be1b PMid:17885550 Matsuo A, Katsuya T, Ishikawa K, Sugimoto K, et al. (2004). G2736A polymorphism of thiazide-sensitive Na-Cl cotransporter gene predisposes to hypertension in young women. J. Hypertens. 22: 2123-2127. doi:10.1097/00004872-200411000-00014 PMid:15480096 Melander O, Orho-Melander M, Bengtsson K, Lindblad U, et al. (2000). Genetic variants of thiazide-sensitive NaCl-cotransporter in Gitelman’s syndrome and primary hypertension. Hypertension 36: 389-394. PMid:10988270 Miao Z, Gao Y, Bindels RJ, Yu W, et al. (2009). Coexistence of normotensive primary aldosteronism in two patients with Gitelman’s syndrome and novel thiazide-sensitive Na-Cl cotransporter mutations. Eur. J. Endocrinol. 161: 275-283. doi:10.1530/EJE-09-0271 PMid:19451210 Obermüller N, Bernstein P, Velazquez H, Reilly R, et al. (1995). Expression of the thiazide-sensitive Na-Cl cotransporter in rat and human kidney. Am. J. Physiol. 269: F900-F910. PMid:8594886 Peters RM and Flack JM (2000). Salt sensitivity and hypertension in African Americans: implications for cardiovascular nurses. Prog. Cardiovasc. Nurs. 15: 138-144. doi:10.1111/j.0889-7204.2000.080404.x PMid:11098526 Plotkin MD, Kaplan MR, Verlander JW, Lee WS, et al. (1996). Localization of the thiazide sensitive Na-Cl cotransporter, rTSC1 in the rat kidney. Kidney Int. 50: 174-183. doi:10.1038/ki.1996.300 PMid:8807586 Shi YY and He L (2005). SHEsis, a powerful software platform for analyses of linkage disequilibrium, haplotype construction, and genetic association at polymorphism loci. Cell Res. 15: 97-98. doi:10.1038/sj.cr.7290272 PMid:15740637 Simon DB, Nelson-Williams C, Bia MJ, Ellison D, et al. (1996). Gitelman’s variant of Bartter’s syndrome, inherited hypokalaemic alkalosis, is caused by mutations in the thiazide-sensitive Na-Cl cotransporter. Nat. Genet. 12: 24-30. doi:10.1038/ng0196-24 PMid:8528245 Slatkin M (2008). Linkage disequilibrium - understanding the evolutionary past and mapping the medical future. Nat. Rev. Genet. 9: 477-485. doi:10.1038/nrg2361 PMid:18427557 Wang XF, Lin RY, Wang SZ, Zhang LP, et al. (2008). Association study of variants in two ion-channel genes (TSC and CLCNKB) and hypertension in two ethnic groups in Northwest China. Clin. Chim. Acta 388: 95-98. doi:10.1016/j.cca.2007.10.017 PMid:17997379 Xiao Z, Xiao J, Jiang Y, Zhang S, et al. (2006). A novel method based on ligase detection reaction for low abundant YIDD mutants detection in hepatitis B virus. Hepatol. Res. 34: 150-155. doi:10.1016/j.hepres.2005.12.007 PMid:16500145 Zhan YY, Jiang X, Lin G, Li J, et al. (2007). Association of thiazide-sensitive Na+-Cl- cotransporter gene polymorphisms with the risk of essential hypertension. Zhonghua Yi. Xue. Yi. Chuan Xue. Za Zhi. 24: 703-705. PMid:18067089
P. Y. Chang, Zhang, X. G., and Su, X. L., “Lack of association of variants of the renal salt reabsorption-related genes SLC12A3 and ClC-Kb and hypertension in Mongolian and Han populations in Inner Mongolia”, vol. 10, pp. 948-954, 2011.
Barlassina C, Dal Fiume C, Lanzani C, Manunta P, et al. (2007). Common genetic variants and haplotypes in renal CLCNKA gene are associated to salt-sensitive hypertension. Hum. Mol. Genet. 16: 1630-1638. doi:10.1093/hmg/ddm112 PMid:17510212 Fava C, Montagnana M, Almgren P, Rosberg L, et al. (2007). The functional variant of the CLC-Kb channel T481S is not associated with blood pressure or hypertension in Swedes. J. Hypertens. 25: 111-116. doi:10.1097/HJH.0b013e3280103a5a PMid:17143181 Guyton AC (1991). Abnormal renal function and autoregulation in essential hypertension. Hypertension 18: III49-III53. PMid:1937686 Jeck N, Waldegger P, Doroszewicz J, Seyberth H, et al. (2004a). A common sequence variation of the CLCNKB gene strongly activates ClC-Kb chloride channel activity. Kidney Int. 65: 190-197. doi:10.1111/j.1523-1755.2004.00363.x PMid:14675050 Jeck N, Waldegger S, Lampert A, Boehmer C, et al. (2004b). Activating mutation of the renal epithelial chloride channel ClC-Kb predisposing to hypertension. Hypertension 43: 1175-1181. doi:10.1161/01.HYP.0000129824.12959.f0 PMid:15148291 Keszei AP, Tisler A, Backx PH, Andrulis IL, et al. (2007). Molecular variants of the thiazide-sensitive Na+-Cl- cotransporter in hypertensive families. J. Hypertens. 25: 2074-2081. doi:10.1097/HJH.0b013e3282a9be1b PMid:17885550 Kokubo Y, Iwai N, Tago N, Inamoto N, et al. (2005). Association analysis between hypertension and CYBA, CLCNKB, and KCNMB1 functional polymorphisms in the Japanese population - the Suita Study. Circ. J. 69: 138-142. doi:10.1253/circj.69.138 PMid:15671602 Lifton RP, Gharavi AG and Geller DS (2001). Molecular mechanisms of human hypertension. Cell 104: 545-556. doi:10.1016/S0092-8674(01)00241-0 Matsuo A, Katsuya T, Ishikawa K, Sugimoto K, et al. (2004). G2736A polymorphism of thiazide-sensitive Na-Cl cotransporter gene predisposes to hypertension in young women. J. Hypertens. 22: 2123-2127. doi:10.1097/00004872-200411000-00014 PMid:15480096 Melander O, Orho-Melander M, Bengtsson K, Lindblad U, et al. (2000). Genetic variants of thiazide-sensitive NaCl-cotransporter in Gitelman’s syndrome and primary hypertension. Hypertension 36: 389-394. PMid:10988270 Obermüller N, Bernstein P, Velázquez H, Reilly R, et al. (1995). Expression of the thiazide-sensitive Na-Cl cotransporter in rat and human kidney. Am. J. Physiol. 269: F900-F910. PMid:8594886 Sile S, Velez DR, Gillani NB, Narsia T, et al. (2009). CLCNKB-T481S and essential hypertension in a Ghanaian population. J. Hypertens. 27: 298-304. doi:10.1097/HJH.0b013e3283140c9e PMid:19226700 Simon DB, Nelson-Williams C, Bia MJ, Ellison D, et al. (1996). Gitelman’s variant of Bartter’s syndrome, inherited hypokalaemic alkalosis, is caused by mutations in the thiazide-sensitive Na-Cl cotransporter. Nat. Genet. 12: 24-30. doi:10.1038/ng0196-24 PMid:8528245 Speirs HJ, Wang WY, Benjafield AV and Morris BJ (2005). No association with hypertension of CLCNKB and TNFRSF1B polymorphisms at a hypertension locus on chromosome 1p36. J. Hypertens. 23: 1491-1496. doi:10.1097/01.hjh.0000174300.73992.cc PMid:16003175 Zhou BF, Wu XG, Tao SQ, Yang J, et al. (1989). Dietary patterns in 10 groups and the relationship with blood pressure. Collaborative Study Group for Cardiovascular Diseases and their Risk Factors. Chin. Med. J. 102: 257-261.