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X. W. Hou, Hu, Z. H., Cui, Y., Gao, W., Sun, S. P., and Wang, N. F., G501C polymorphism of the oxidized LDL receptor gene is associated with albuminuria in Chinese essential hypertension patients, vol. 10, pp. 2710-2717, 2011.
Basi S and Lewis JB (2006). Microalbuminuria as a target to improve cardiovascular and renal outcomes. Am J. Kidney Dis. 47: 927-946. S, Falconi M, Filesi I, Baldini F, et al. (2009). Functional analysis and molecular dynamics simulation of LOX-1 K167N polymorphism reveal alteration of receptor activity. PLoS One 4: e4648.    PMCid:2645694Brinkley TE, Kume N, Mitsuoka H, Brown MD, et al. (2008). Variation in the human lectin-like oxidized low-density lipoprotein receptor 1 (LOX-1) gene is associated with plasma soluble LOX-1 levels. Exp. Physiol. 93: 1085-1090.    PMCid:2652129Cerasola G, Mule G, Cottone S, Nardi E, et al. (2008). Hypertension, microalbuminuria and renal dysfunction: the Renal Dysfunction in Hypertension (REDHY) study. J. Nephrol. 21: 368-373.PMid:18587725Cilingiroglu M and Ozer K (2005). The lectin-like oxidized low-density lipoprotein receptor and its role in atherosclerosis. Curr. Atheroscler. Rep. 7: 103-107. L, Fratta PA, Garbin U, Pastorino A, et al. (2003). The platelet-endothelium interaction mediated by lectin-like oxidized low-density lipoprotein receptor-1 reduces the intracellular concentration of nitric oxide in endothelial cells. J. Am. Coll. Cardiol. 41: 499-507. LM, Schwartz JE and Pickering TG (2006). Albumin-to-creatinine ratio predicts change in ambulatory blood pressure in normotensive persons: a 7.5-year prospective study. Am. J. Hypertens. 19: 220-226. H, Sonoda A, Sato H, Ito D, et al. (2006). G501C polymorphism of oxidized LDL receptor gene (OLR1) and ischemic stroke. Brain Res. 1121: 246-249. XW, Wang LF, Wang N, Pang D, et al. (2008). The G501C polymorphism of oxidized LDL receptor gene [OLR-1] is associated with susceptibility and serum C-reactive protein concentration in Chinese essential hypertensives. Clin. Chim. Acta 388: 200-203. C, Kang BY, Megyesi J, Kaushal GP, et al. (2009). Deletion of LOX-1 attenuates renal injury following angiotensin II infusion. Kidney Int. 76: 521-527. KP, Parving HH, Scharling H and Jensen JS (2007). The association between metabolic syndrome, microalbuminuria and impaired renal function in the general population: impact on cardiovascular disease and mortality. J. Intern. Med. 262: 470-478. KP, Parving HH, Scharling H and Jensen JS (2009). Microalbuminuria and obesity: impact on cardiovascular disease and mortality. Clin. Endocrinol. 71: 40-45. O, Aydogan HY, Isbir CS, Tekeli A, et al. (2009). Is LOX-1 K167N polymorphism protective for coronary artery disease? In Vivo 23: 969-973.PMid:20023241Li L, Roumeliotis N, Sawamura T and Renier G (2004). C-reactive protein enhances LOX-1 expression in human aortic endothelial cells: relevance of LOX-1 to C-reactive protein-induced endothelial dysfunction. Circ. Res. 95: 877-883. R, Biocca S, del VF, Clementi F, et al. (2005). In vivo and in vitro studies support that a new splicing isoform of OLR1 gene is protective against acute myocardial infarction. Circ. Res. 97: 152-158. M, Kaname S, Nagase T, Wang G, et al. (2000). Expression of LOX-1, an oxidized low-density lipoprotein receptor, in experimental hypertensive glomerulosclerosis. J. Am. Soc. Nephrol. 11: 1826-1836.PMid:11004213Ogura S, Kakino A, Sato Y, Fujita Y, et al. (2009). Lox-1: the multifunctional receptor underlying cardiovascular dysfunction. Circ. J. 73: 1993-1999. R, Momiyama Y, Nagano M, Taniguchi H, et al. (2004). An oxidized low-density lipoprotein receptor gene variant is inversely associated with the severity of coronary artery disease. Clin. Cardiol. 27: 641-644. F, Maio R, Tripepi G, Sciacqua A, et al. (2007). Microalbuminuria, endothelial dysfunction and inflammation in primary hypertension. J. Nephrol. 20 (Suppl 12): S56-S62.PMid:18050145Perticone F, Maio R, Perticone M, Sciacqua A, et al. (2010). Endothelial dysfunction and subsequent decline in glomerular filtration rate in hypertensive patients. Circulation 122: 379-384. J, Luders S, Kulschewski A, Hammersen F, et al. (2006). Microalbuminuria and tubular proteinuria as risk predictors of cardiovascular morbidity and mortality in essential hypertension: final results of a prospective long-term study (MARPLE Study)*. J. Hypertens. 24: 541-548. M, Furutani M, Hinagata J, Tanaka T, et al. (2003). Oxidized LDL receptor gene (OLR1) is associated with the risk of myocardial infarction. Biochem. Biophys. Res. Commun. 303: 247-250. E, Biscuola M, Cavallari U, Malerba G, et al. (2006). On the association of the oxidised LDL receptor 1 (OLR1) gene in patients with acute myocardial infarction or coronary artery disease. Eur. J. Hum. Genet. 14: 127-130.PMid:16251892Tsioufis C, Dimitriadis K, Andrikou E, Thomopoulos C, et al. (2010). ADMA, C-reactive protein, and albuminuria in untreated essential hypertension: a cross-sectional study. Am. J. Kidney Dis. 55: 1050-1059. T, Kaname S, Takaichi K, Nagase M, et al. (2003). LOX-1, an oxidized low-density lipoprotein receptor, was upregulated in the kidneys of chronic renal failure rats. Hypertens Res. 26: 117-122. T, Fukuda N, Tsunemi A, Yao EH, et al. (2009). A novel gene silencer, pyrrole-imidazole polyamide targeting human lectin-like oxidized low-density lipoprotein receptor-1 gene improves endothelial cell function. J. Hypertens. 27: 508-516. F, Leoncini G, Conti N, Tomolillo C, et al. (2010). Microalbuminuria is a predictor of chronic renal insufficiency in patients without diabetes and with hypertension: the MAGIC study. Clin. J. Am. Soc. Nephrol. 5: 1099-1106.    PMCid:2879305Yamamoto N, Toyoda M, Abe M, Kobayashi T, et al. (2009). Lectin-like oxidized LDL receptor-1 (LOX-1) expression in the tubulointerstitial area likely plays an important role in human diabetic nephropathy. Intern. Med. 48: 189-194. MF, Khaw KT, Luben R, Welch A, et al. (2004). Microalbuminuria independently predicts all-cause and cardiovascular mortality in a British population: The European Prospective Investigation into Cancer in Norfolk (EPIC-Norfolk) population study. Int. J. Epidemiol. 33: 189-198.