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2013
Y. H. Ling, Ding, J. P., Zhang, X. D., Wang, L. J., Zhang, Y. H., Li, Y. S., Zhang, Z. J., and Zhang, X. R., Characterization of microRNAs from goat (Capra hircus) by Solexa deep-sequencing technology, vol. 12, pp. 1951-1961, 2013.
Y. Z. Li, Wang, L. J., Li, X., Li, S. L., Wang, J. L., Wu, Z. H., Gong, L., and Zhang, X. D., Vascular endothelial growth factor gene polymorphisms contribute to the risk of endometriosis: an updated systematic review and meta-analysis of 14 case-control studies, vol. 12, pp. 1035-1044, 2013.
Altinkaya SO, Ugur M, Ceylaner G, Ozat M, et al. (2011). Vascular endothelial growth factor +405 C/G polymorphism is highly associated with an increased risk of endometriosis in Turkish women. Arch. Gynecol. Obstet. 283: 267-272. http://dx.doi.org/10.1007/s00404-009-1344-1 PMid:20041256   Attar R, Agachan B, Kuran SB, Toptas B, et al. (2010). Genetic variants of vascular endothelial growth factor and risk for the development of endometriosis. In Vivo 24: 297-301. PMid:20555002   Bhanoori M, Arvind BK, Pavankumar Reddy NG, Lakshmi RK, et al. (2005). The vascular endothelial growth factor (VEGF) +405G>C 5'-untranslated region polymorphism and increased risk of endometriosis in South Indian women: a case control study. Hum. Reprod. 20: 1844-1849. http://dx.doi.org/10.1093/humrep/deh852 PMid:15746194   Cosin R, Gilabert-Estelles J, Ramon LA, Espana F, et al. (2009). Vascular endothelial growth factor polymorphisms (-460C/T, +405G/C, and 936C/T) and endometriosis: their influence on vascular endothelial growth factor expression. Fertil. Steril. 92: 1214-1220. http://dx.doi.org/10.1016/j.fertnstert.2008.08.079 PMid:18930211   Ferrara N (2004). Vascular endothelial growth factor: basic science and clinical progress. Endocr. Rev. 25: 581-611. http://dx.doi.org/10.1210/er.2003-0027 PMid:15294883   Ferrara N, Gerber HP and LeCouter J (2003). The biology of VEGF and its receptors. Nat. Med. 9: 669-676. http://dx.doi.org/10.1038/nm0603-669 PMid:12778165   Fukumura D, Xavier R, Sugiura T, Chen Y, et al. (1998). Tumor induction of VEGF promoter activity in stromal cells. Cell 94: 715-725. http://dx.doi.org/10.1016/S0092-8674(00)81731-6   Gentilini D, Somigliana E, Vigano P, Vignali M, et al. (2008). The vascular endothelial growth factor +405G>C polymorphism in endometriosis. Hum. Reprod. 23: 211-215. http://dx.doi.org/10.1093/humrep/dem341 PMid:17977866   Girling JE and Rogers PA (2005). Recent advances in endometrial angiogenesis research. Angiogenesis 8: 89-99. http://dx.doi.org/10.1007/s10456-005-9006-9 PMid:16211359   Higgins JP and Thompson SG (2002). Quantifying heterogeneity in a meta-analysis. Stat. Med. 21: 1539-1558. http://dx.doi.org/10.1002/sim.1186 PMid:12111919   Hsieh YY, Chang CC, Tsai FJ, Yeh LS, et al. (2004). T allele for VEGF gene-460 polymorphism at the 5'-untranslated region: association with a higher susceptibility to endometriosis. J. Reprod. Med. 49: 468-472. PMid:15283056   Ikuhashi Y, Yoshida S, Kennedy S, Zondervan K, et al. (2007). Vascular endothelial growth factor +936 C/T polymorphism is associated with an increased risk of endometriosis in a Japanese population. Acta Obstet. Gynecol. Scand. 86: 1352-1358. http://dx.doi.org/10.1080/00016340701644991 PMid:17963063   Kang S, Zhao J, Liu Q, Zhou R, et al. (2009). Vascular endothelial growth factor gene polymorphisms are associated with the risk of developing adenomyosis. Environ. Mol. Mutagen. 50: 361-366. http://dx.doi.org/10.1002/em.20455 PMid:19197986   Kim JG, Kim JY, Jee BC, Suh CS, et al. (2008). Association between endometriosis and polymorphisms in endostatin and vascular endothelial growth factor and their serum levels in Korean women. Fertil. Steril. 89: 243-245. http://dx.doi.org/10.1016/j.fertnstert.2007.02.023 PMid:17482599   Kim SH, Choi YM, Choung SH, Jun JK, et al. (2005). Vascular endothelial growth factor gene +405 C/G polymorphism is associated with susceptibility to advanced stage endometriosis. Hum. Reprod. 20: 2904-2908. http://dx.doi.org/10.1093/humrep/dei146 PMid:15979997   Lamp M, Saare M, Laisk T, Karro H, et al. (2010). Genetic variations in vascular endothelial growth factor but not in angiotensin I-converting enzyme genes are associated with endometriosis in Estonian women. Eur. J. Obstet. Gynecol. Reprod. Biol. 153: 85-89. http://dx.doi.org/10.1016/j.ejogrb.2010.07.021 PMid:20685027   Liu Q, Li Y, Zhao J, Sun DL, et al. (2009a). Association of polymorphisms -1154G/A and -2578C/A in the vascular endothelial growth factor gene with decreased risk of endometriosis in Chinese women. Hum. Reprod. 24: 2660-2666. http://dx.doi.org/10.1093/humrep/dep208 PMid:19531502   Liu Q, Li Y, Zhao J, Zhou RM, et al. (2009b). Association of single nucleotide polymorphisms in VEGF gene with the risk of endometriosis and adenomyosis. Zhonghua Yi Xue Yi Chuan Xue Za Zhi 26: 165-169. PMid:19350508   Matalliotakis IM, Katsikis IK and Panidis DK (2005). Adenomyosis: what is the impact on fertility? Curr. Opin. Obstet. Gynecol. 17: 261-264. http://dx.doi.org/10.1097/01.gco.0000169103.85128.c0 PMid:15870560   Missmer SA and Cramer DW (2003). The epidemiology of endometriosis. Obstet. Gynecol. Clin. North Am. 30: 1-19, vii. http://dx.doi.org/10.1016/S0889-8545(02)00050-5   Peters JL, Sutton AJ, Jones DR, Abrams KR, et al. (2006). Comparison of two methods to detect publication bias in meta-analysis. JAMA 295: 676-680. http://dx.doi.org/10.1001/jama.295.6.676 PMid:16467236   Signorile PG and Baldi A (2010). Endometriosis: new concepts in the pathogenesis. Int. J. Biochem. Cell Biol. 42: 778-780. http://dx.doi.org/10.1016/j.biocel.2010.03.008 PMid:20230903   Varma R, Rollason T, Gupta JK and Maher ER (2004). Endometriosis and the neoplastic process. Reproduction 127: 293-304. http://dx.doi.org/10.1530/rep.1.00020 PMid:15016949   Vigano P, Parazzini F, Somigliana E and Vercellini P (2004). Endometriosis: epidemiology and aetiological factors. Best. Pract. Res. Clin. Obstet. Gynaecol. 18: 177-200. http://dx.doi.org/10.1016/j.bpobgyn.2004.01.007 PMid:15157637   von Elm E, Altman DG, Egger M, Pocock SJ, et al. (2007). The Strengthening the Reporting of Observational Studies in Epidemiology (STROBE) statement: guidelines for reporting observational studies. Lancet 370: 1453-1457. http://dx.doi.org/10.1016/S0140-6736(07)61602-X   Watson CJ, Webb NJ, Bottomley MJ and Brenchley PE (2000). Identification of polymorphisms within the vascular endothelial growth factor (VEGF) gene: correlation with variation in VEGF protein production. Cytokine 12: 1232- 1235. http://dx.doi.org/10.1006/cyto.2000.0692 PMid:10930302   Zhang L, Liu JL, Zhang YJ and Wang H (2011). Association between HLA-B*27 polymorphisms and ankylosing spondylitis in Han populations: a meta-analysis. Clin. Exp. Rheumatol 29: 285-292. PMid:21418777   Zhao ZZ, Nyholt DR, Thomas S, Treloar SA, et al. (2008). Polymorphisms in the vascular endothelial growth factor gene and the risk of familial endometriosis. Mol. Hum. Reprod. 14: 531-538. http://dx.doi.org/10.1093/molehr/gan043 PMid:18650217   Zintzaras E and Ioannidis JP (2005). Heterogeneity testing in meta-analysis of genome searches. Genet. Epidemiol. 28: 123-137. http://dx.doi.org/10.1002/gepi.20048 PMid:15593093
2011
D. Wu, Wu, Y., Liu, J. L., Wang, B., and Zhang, X. D., Association between HLA-Cw*0602 polymorphism and psoriasis risk: a meta-analysis, vol. 10, pp. 3109-3120, 2011.
Asumalahti K, Laitinen T, Itkonen-Vatjus R, Lokki ML, et al. (2000). A candidate gene for psoriasis near HLA-C, HCR (Pg8), is highly polymorphic with a disease-associated susceptibility allele. Hum. Mol. Genet. 9: 1533-1542. http://dx.doi.org/10.1093/hmg/9.10.1533 PMid:10888604 Attia J, Thakkinstian A and D’Este C (2003). Meta-analyses of molecular association studies: methodologic lessons for genetic epidemiology. J. Clin. Epidemiol. 56: 297-303. http://dx.doi.org/10.1016/S0895-4356(03)00011-8 Brandrup F, Holm N, Grunnet N, Henningsen K, et al. (1982). Psoriasis in monozygotic twins: variations in expression in individuals with identical genetic constitution. Acta Derm. Venereol. 62: 229-236. PMid:6179364 Brazzelli V, Quaglini M, Martinetti M, Nolli G, et al. (2000). A peculiar sequence motif in the alpha-1-domain of the HLA-C molecule in psoriasis. Dermatology 200: 99-103. http://dx.doi.org/10.1159/000018338 PMid:10773694 Chandran V and Raychaudhuri SP (2010). Geoepidemiology and environmental factors of psoriasis and psoriatic arthritis. J. Autoimmun. 34: J314-J321. http://dx.doi.org/10.1016/j.jaut.2009.12.001 PMid:20034760 Chang YT, Shiao YM, Chin PJ, Liu YL, et al. (2004). Genetic polymorphisms of the HCR gene and a genomic segment in close proximity to HLA-C are associated with patients with psoriasis in Taiwan. Br. J. Dermatol. 150: 1104-1111. http://dx.doi.org/10.1111/j.1365-2133.2004.05972.x PMid:15214895 Chang YT, Liu HN, Shiao YM, Lin MW, et al. (2005). A study of PSORS1C1 gene polymorphisms in Chinese patients with psoriasis. Br. J. Dermatol. 153: 90-96. http://dx.doi.org/10.1111/j.1365-2133.2005.06570.x PMid:16029332 Chang YT, Chou CT, Shiao YM, Lin MW, et al. (2006). Psoriasis vulgaris in Chinese individuals is associated with PSORS1C3 and CDSN genes. Br. J. Dermatol. 155: 663-669. http://dx.doi.org/10.1111/j.1365-2133.2006.07420.x PMid:16965413 Duffin KC, Chandran V, Gladman DD, Krueger GG, et al. (2008). Genetics of psoriasis and psoriatic arthritis: update and future direction. J. Rheumatol. 35: 1449-1453. PMid:18609743    PMCid:2724000 Fan X, Yang S, Sun LD, Liang YH, et al. (2007). Comparison of clinical features of HLA-Cw*0602-positive and -negative psoriasis patients in a Han Chinese population. Acta Derm. Venereol. 87: 335-340. http://dx.doi.org/10.2340/00015555-0253 PMid:17598037 Farber EM, Nall ML and Watson W (1974). Natural history of psoriasis in 61 twin pairs. Arch Dermatol. 109: 207-211. http://dx.doi.org/10.1001/archderm.1974.01630020023005 PMid:4814926 Fojtíková M, Stolfa J, Novota P, Cejkova P, et al. (2009). HLA-Cw*06 class I region rather than MICA is associated with psoriatic arthritis in Czech population. Rheumatol. Int. 29: 1293-1299. http://dx.doi.org/10.1007/s00296-009-0847-1 PMid:19184033 Gonzalez S, Martinez-Borra J, Torre-Alonso JC, Gonzalez-Roces S, et al. (1999). The MICA-A9 triplet repeat polymorphism in the transmembrane region confers additional susceptibility to the development of psoriatic arthritis and is independent of the association of Cw*0602 in psoriasis. Arthritis Rheum. 42: 1010-1016. http://dx.doi.org/10.1002/1529-0131(199905)42:5<1010::AID-ANR21>3.0.CO;2-H Griffiths CE and Barker JN (2007). Pathogenesis and clinical features of psoriasis. Lancet 370: 263-271. http://dx.doi.org/10.1016/S0140-6736(07)61128-3 Gudjonsson JE, Karason A, Antonsdottir A, Runarsdottir EH, et al. (2003). Psoriasis patients who are homozygous for the HLA-Cw*0602 allele have a 2.5-fold increased risk of developing psoriasis compared with Cw6 heterozygotes. Br. J. Dermatol. 148: 233-235. http://dx.doi.org/10.1046/j.1365-2133.2003.05115.x PMid:12588373 Gudjonsson JE, Johnston A, Sigmundsdottir H and Valdimarsson H (2004). Immunopathogenic mechanisms in psoriasis. Clin. Exp. Immunol. 135: 1-8. http://dx.doi.org/10.1111/j.1365-2249.2004.02310.x PMid:14678257    PMCid:1808928 Higgins JP and Thompson SG (2002). Quantifying heterogeneity in a meta-analysis. Stat. Med. 21: 1539-1558. http://dx.doi.org/10.1002/sim.1186 PMid:12111919 Holm SJ, Sanchez F, Carlen LM, Mallbris L, et al. (2005a). HLA-Cw*0602 associates more strongly to psoriasis in the Swedish population than variants of the novel 6p21.3 gene PSORS1C3. Acta Derm. Venereol. 85: 2-8. http://dx.doi.org/10.1080/00015550410023527 Holm SJ, Sakuraba K, Mallbris L, Wolk K, et al. (2005b). Distinct HLA-C/KIR genotype profile associates with guttate psoriasis. J. Invest. Dermatol. 125: 721-730. http://dx.doi.org/10.1111/j.0022-202X.2005.23879.x PMid:16185272 Jobim M, Jobim LF, Salim PH, Cestari TF, et al. (2008). A study of the killer cell immunoglobulin-like receptor gene KIR2DS1 in a Caucasoid Brazilian population with Psoriasis vulgaris. Tissue Antigens 72: 392-396. http://dx.doi.org/10.1111/j.1399-0039.2008.01096.x PMid:18643961 Liu Y, Helms C, Liao W, Zaba LC, et al. (2008). A genome-wide association study of psoriasis and psoriatic arthritis identifies new disease loci. PLoS Genet. 4: e1000041. http://dx.doi.org/10.1371/journal.pgen.1000041 PMid:18369459    PMCid:2274885 Lowes MA, Bowcock AM and Krueger JG (2007). Pathogenesis and therapy of psoriasis. Nature 445: 866-873. http://dx.doi.org/10.1038/nature05663 PMid:17314973 Luft FC (2005). Light shed on the common skin scourge, psoriasis. J. Mol. Med. 83: 933-934. http://dx.doi.org/10.1007/s00109-005-0724-7 PMid:16323032 Luszczek W, Kubicka W, Cislo M, Nockowski P, et al. (2003). Strong association of HLA-Cw6 allele with juvenile psoriasis in Polish patients. Immunol. Lett. 85: 59-64. http://dx.doi.org/10.1016/S0165-2478(02)00212-2 Mallbris L, Wolk K, Sanchez F and Stahle M (2009). HLA-Cw*0602 associates with a twofold higher prevalence of positive streptococcal throat swab at the onset of psoriasis: a case control study. BMC Dermatol. 9: 5. http://dx.doi.org/10.1186/1471-5945-9-5 PMid:19480679    PMCid:2696405 Martínez-Borra J, Gonzalez S, Santos-Juanes J, Sanchez del RJ, et al. (2003). Psoriasis vulgaris and psoriatic arthritis share a 100 kb susceptibility region telomeric to HLA-C. Rheumatology 42: 1089-1092. http://dx.doi.org/10.1093/rheumatology/keg304 PMid:12730526 Martínez-Borra J, Brautbar C, Gonzalez S, Enk CD, et al. (2005). The region of 150 kb telometic to HLA-C is associated with psoriasis in the Jewish population. J. Invest. Dermatol. 125: 928-932. PMid:16297191 Nair RP, Stuart PE, Nistor I, Hiremagalore R, et al. (2006). Sequence and haplotype analysis supports HLA-C as the psoriasis susceptibility 1 gene. Am. J. Hum. Genet. 78: 827-851. http://dx.doi.org/10.1086/503821 PMid:16642438 Naldi L and Mercuri SR (2010). Epidemiology of comorbidities in psoriasis. Dermatol. Ther. 23: 114-118. http://dx.doi.org/10.1111/j.1529-8019.2010.01304.x PMid:20415817 Nestle FO, Kaplan DH and Barker J (2009). Psoriasis. N. Engl. J. Med. 361: 496-509. http://dx.doi.org/10.1056/NEJMra0804595 PMid:19641206 Peters JL, Sutton AJ, Jones DR, Abrams KR, et al. (2006). Comparison of two methods to detect publication bias in meta-analysis. JAMA 295: 676-680. http://dx.doi.org/10.1001/jama.295.6.676 PMid:16467236 Płoski R, Luszczek W, Kuśnierczyk P, Nockowski P, et al. (2006). A role for KIR gene variants other than KIR2DS1 in conferring susceptibility to psoriasis. Hum. Immunol. 67: 521-526. http://dx.doi.org/10.1016/j.humimm.2006.04.001 PMid:16829306 Rani R, Narayan R, Fernandez-Vina MA and Stastny P (1998). Role of HLA-B and C alleles in development of psoriasis in patients from North India. Tissue Antigens 51: 618-622. http://dx.doi.org/10.1111/j.1399-0039.1998.tb03004.x PMid:9694354 Raychaudhuri SP and Gross J (2000a). A comparative study of pediatric onset psoriasis with adult onset psoriasis. Pediatr. Dermatol. 17: 174-178. http://dx.doi.org/10.1046/j.1525-1470.2000.01746.x PMid:10886746 Raychaudhuri SP and Gross J (2000b). Psoriasis risk factors: role of lifestyle practices. Cutis 66: 348-352. PMid:11107520 Raychaudhuri SP and Farber EM (2001). The prevalence of psoriasis in the world. J. Eur. Acad. Dermatol. Venereol. 15: 16-17. http://dx.doi.org/10.1046/j.1468-3083.2001.00192.x PMid:11451313 Reveille JD and Williams FM (2006). Infection and musculoskeletal conditions: Rheumatologic complications of HIV infection. Best Pract. Res. Clin. Rheumatol. 20: 1159-1179. http://dx.doi.org/10.1016/j.berh.2006.08.015 Sánchez F, Holm SJ, Mallbris L, O’Brien KP, et al. (2004). STG does not associate with psoriasis in the Swedish population. Exp. Dermatol. 13: 413-418. http://dx.doi.org/10.1111/j.0906-6705.2004.00170.x PMid:15217361 Schon MP and Boehncke WH (2005). Psoriasis. N. Engl. J. Med. 352: 1899-1912. http://dx.doi.org/10.1056/NEJMra041320 PMid:15872205 Szczerkowska-Dobosz A (2005). Human leukocyte antigens as psoriasis inheritance and susceptibility markers. Arch. Immunol. Ther. Exp. 53: 428-433. Valdimarsson H (2007). The genetic basis of psoriasis. Clin. Dermatol. 25: 563-567. http://dx.doi.org/10.1016/j.clindermatol.2007.08.010 PMid:18021893 Vandenbroucke JP, von Elm E, Altman DG, Gotzsche PC, et al. (2007). Strengthening the Reporting of Observational Studies in Epidemiology (STROBE): explanation and elaboration. Epidemiology 18: 805-835. http://dx.doi.org/10.1097/EDE.0b013e3181577511 PMid:18049195 Watson W, Cann HM, Farber EM and Nall ML (1972). The genetics of psoriasis. Arch. Dermatol. 105: 197-207. http://dx.doi.org/10.1001/archderm.1972.01620050011002 PMid:5060862 Zhang XJ, Zhang AP, Yang S, Gao M, et al. (2003). Association of HLA class I alleles with Psoriasis vulgaris in southeastern Chinese Hans. J. Dermatol. Sci. 33: 1-6. http://dx.doi.org/10.1016/S0923-1811(03)00157-9 Zintzaras E and Ioannidis JP (2005). Heterogeneity testing in meta-analysis of genome searches. Genet. Epidemiol. 28: 123-137. http://dx.doi.org/10.1002/gepi.20048 PMid:15593093