Publications
Found 9 results
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“Analysis of skin color change and related gene expression after crossing of Dongxiang black chicken and ISA layer”, vol. 14, pp. 11551-11561, 2015.
, “Association between ADSL, GARS-AIRS-GART, DGAT1, and DECR1 expression levels and pork meat quality traits”, vol. 14, pp. 14823-14830, 2015.
, “Expression of OPG, RANKL, and RUNX2 in rabbit periodontium under orthodontic force”, vol. 14, pp. 19382-19388, 2015.
, “High-resolution melting curve analysis of the ADSL and LPL genes and their correlation with meat quality and blood parameters in chickens”, vol. 14, pp. 2031-2040, 2015.
, “Relationship between polymorphisms in exon 10 of FSHR gene and litter size in swine”, vol. 14, pp. 8252-8261, 2015.
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“Characterization of microRNAs from goat (Capra hircus) by Solexa deep-sequencing technology”, vol. 12, pp. 1951-1961, 2013.
, “Vascular endothelial growth factor gene polymorphisms contribute to the risk of endometriosis: an updated systematic review and meta-analysis of 14 case-control studies”, vol. 12, pp. 1035-1044, 2013.
, Altinkaya SO, Ugur M, Ceylaner G, Ozat M, et al. (2011). Vascular endothelial growth factor +405 C/G polymorphism is highly associated with an increased risk of endometriosis in Turkish women. Arch. Gynecol. Obstet. 283: 267-272.
http://dx.doi.org/10.1007/s00404-009-1344-1
PMid:20041256
Attar R, Agachan B, Kuran SB, Toptas B, et al. (2010). Genetic variants of vascular endothelial growth factor and risk for the development of endometriosis. In Vivo 24: 297-301.
PMid:20555002
Bhanoori M, Arvind BK, Pavankumar Reddy NG, Lakshmi RK, et al. (2005). The vascular endothelial growth factor (VEGF) +405G>C 5'-untranslated region polymorphism and increased risk of endometriosis in South Indian women: a case control study. Hum. Reprod. 20: 1844-1849.
http://dx.doi.org/10.1093/humrep/deh852
PMid:15746194
Cosin R, Gilabert-Estelles J, Ramon LA, Espana F, et al. (2009). Vascular endothelial growth factor polymorphisms (-460C/T, +405G/C, and 936C/T) and endometriosis: their influence on vascular endothelial growth factor expression. Fertil. Steril. 92: 1214-1220.
http://dx.doi.org/10.1016/j.fertnstert.2008.08.079
PMid:18930211
Ferrara N (2004). Vascular endothelial growth factor: basic science and clinical progress. Endocr. Rev. 25: 581-611.
http://dx.doi.org/10.1210/er.2003-0027
PMid:15294883
Ferrara N, Gerber HP and LeCouter J (2003). The biology of VEGF and its receptors. Nat. Med. 9: 669-676.
http://dx.doi.org/10.1038/nm0603-669
PMid:12778165
Fukumura D, Xavier R, Sugiura T, Chen Y, et al. (1998). Tumor induction of VEGF promoter activity in stromal cells. Cell 94: 715-725.
http://dx.doi.org/10.1016/S0092-8674(00)81731-6
Gentilini D, Somigliana E, Vigano P, Vignali M, et al. (2008). The vascular endothelial growth factor +405G>C polymorphism in endometriosis. Hum. Reprod. 23: 211-215.
http://dx.doi.org/10.1093/humrep/dem341
PMid:17977866
Girling JE and Rogers PA (2005). Recent advances in endometrial angiogenesis research. Angiogenesis 8: 89-99.
http://dx.doi.org/10.1007/s10456-005-9006-9
PMid:16211359
Higgins JP and Thompson SG (2002). Quantifying heterogeneity in a meta-analysis. Stat. Med. 21: 1539-1558.
http://dx.doi.org/10.1002/sim.1186
PMid:12111919
Hsieh YY, Chang CC, Tsai FJ, Yeh LS, et al. (2004). T allele for VEGF gene-460 polymorphism at the 5'-untranslated region: association with a higher susceptibility to endometriosis. J. Reprod. Med. 49: 468-472.
PMid:15283056
Ikuhashi Y, Yoshida S, Kennedy S, Zondervan K, et al. (2007). Vascular endothelial growth factor +936 C/T polymorphism is associated with an increased risk of endometriosis in a Japanese population. Acta Obstet. Gynecol. Scand. 86: 1352-1358.
http://dx.doi.org/10.1080/00016340701644991
PMid:17963063
Kang S, Zhao J, Liu Q, Zhou R, et al. (2009). Vascular endothelial growth factor gene polymorphisms are associated with the risk of developing adenomyosis. Environ. Mol. Mutagen. 50: 361-366.
http://dx.doi.org/10.1002/em.20455
PMid:19197986
Kim JG, Kim JY, Jee BC, Suh CS, et al. (2008). Association between endometriosis and polymorphisms in endostatin and vascular endothelial growth factor and their serum levels in Korean women. Fertil. Steril. 89: 243-245.
http://dx.doi.org/10.1016/j.fertnstert.2007.02.023
PMid:17482599
Kim SH, Choi YM, Choung SH, Jun JK, et al. (2005). Vascular endothelial growth factor gene +405 C/G polymorphism is associated with susceptibility to advanced stage endometriosis. Hum. Reprod. 20: 2904-2908.
http://dx.doi.org/10.1093/humrep/dei146
PMid:15979997
Lamp M, Saare M, Laisk T, Karro H, et al. (2010). Genetic variations in vascular endothelial growth factor but not in angiotensin I-converting enzyme genes are associated with endometriosis in Estonian women. Eur. J. Obstet. Gynecol. Reprod. Biol. 153: 85-89.
http://dx.doi.org/10.1016/j.ejogrb.2010.07.021
PMid:20685027
Liu Q, Li Y, Zhao J, Sun DL, et al. (2009a). Association of polymorphisms -1154G/A and -2578C/A in the vascular endothelial growth factor gene with decreased risk of endometriosis in Chinese women. Hum. Reprod. 24: 2660-2666.
http://dx.doi.org/10.1093/humrep/dep208
PMid:19531502
Liu Q, Li Y, Zhao J, Zhou RM, et al. (2009b). Association of single nucleotide polymorphisms in VEGF gene with the risk of endometriosis and adenomyosis. Zhonghua Yi Xue Yi Chuan Xue Za Zhi 26: 165-169.
PMid:19350508
Matalliotakis IM, Katsikis IK and Panidis DK (2005). Adenomyosis: what is the impact on fertility? Curr. Opin. Obstet. Gynecol. 17: 261-264.
http://dx.doi.org/10.1097/01.gco.0000169103.85128.c0
PMid:15870560
Missmer SA and Cramer DW (2003). The epidemiology of endometriosis. Obstet. Gynecol. Clin. North Am. 30: 1-19, vii.
http://dx.doi.org/10.1016/S0889-8545(02)00050-5
Peters JL, Sutton AJ, Jones DR, Abrams KR, et al. (2006). Comparison of two methods to detect publication bias in meta-analysis. JAMA 295: 676-680.
http://dx.doi.org/10.1001/jama.295.6.676
PMid:16467236
Signorile PG and Baldi A (2010). Endometriosis: new concepts in the pathogenesis. Int. J. Biochem. Cell Biol. 42: 778-780.
http://dx.doi.org/10.1016/j.biocel.2010.03.008
PMid:20230903
Varma R, Rollason T, Gupta JK and Maher ER (2004). Endometriosis and the neoplastic process. Reproduction 127: 293-304.
http://dx.doi.org/10.1530/rep.1.00020
PMid:15016949
Vigano P, Parazzini F, Somigliana E and Vercellini P (2004). Endometriosis: epidemiology and aetiological factors. Best. Pract. Res. Clin. Obstet. Gynaecol. 18: 177-200.
http://dx.doi.org/10.1016/j.bpobgyn.2004.01.007
PMid:15157637
von Elm E, Altman DG, Egger M, Pocock SJ, et al. (2007). The Strengthening the Reporting of Observational Studies in Epidemiology (STROBE) statement: guidelines for reporting observational studies. Lancet 370: 1453-1457.
http://dx.doi.org/10.1016/S0140-6736(07)61602-X
Watson CJ, Webb NJ, Bottomley MJ and Brenchley PE (2000). Identification of polymorphisms within the vascular endothelial growth factor (VEGF) gene: correlation with variation in VEGF protein production. Cytokine 12: 1232- 1235.
http://dx.doi.org/10.1006/cyto.2000.0692
PMid:10930302
Zhang L, Liu JL, Zhang YJ and Wang H (2011). Association between HLA-B*27 polymorphisms and ankylosing spondylitis in Han populations: a meta-analysis. Clin. Exp. Rheumatol 29: 285-292.
PMid:21418777
Zhao ZZ, Nyholt DR, Thomas S, Treloar SA, et al. (2008). Polymorphisms in the vascular endothelial growth factor gene and the risk of familial endometriosis. Mol. Hum. Reprod. 14: 531-538.
http://dx.doi.org/10.1093/molehr/gan043
PMid:18650217
Zintzaras E and Ioannidis JP (2005). Heterogeneity testing in meta-analysis of genome searches. Genet. Epidemiol. 28: 123-137.
http://dx.doi.org/10.1002/gepi.20048
PMid:15593093
“Association between HLA-Cw*0602 polymorphism and psoriasis risk: a meta-analysis”, vol. 10, pp. 3109-3120, 2011.
, Asumalahti K, Laitinen T, Itkonen-Vatjus R, Lokki ML, et al. (2000). A candidate gene for psoriasis near HLA-C, HCR (Pg8), is highly polymorphic with a disease-associated susceptibility allele. Hum. Mol. Genet. 9: 1533-1542.
http://dx.doi.org/10.1093/hmg/9.10.1533
PMid:10888604
Attia J, Thakkinstian A and D’Este C (2003). Meta-analyses of molecular association studies: methodologic lessons for genetic epidemiology. J. Clin. Epidemiol. 56: 297-303.
http://dx.doi.org/10.1016/S0895-4356(03)00011-8
Brandrup F, Holm N, Grunnet N, Henningsen K, et al. (1982). Psoriasis in monozygotic twins: variations in expression in individuals with identical genetic constitution. Acta Derm. Venereol. 62: 229-236.
PMid:6179364
Brazzelli V, Quaglini M, Martinetti M, Nolli G, et al. (2000). A peculiar sequence motif in the alpha-1-domain of the HLA-C molecule in psoriasis. Dermatology 200: 99-103.
http://dx.doi.org/10.1159/000018338
PMid:10773694
Chandran V and Raychaudhuri SP (2010). Geoepidemiology and environmental factors of psoriasis and psoriatic arthritis. J. Autoimmun. 34: J314-J321.
http://dx.doi.org/10.1016/j.jaut.2009.12.001
PMid:20034760
Chang YT, Shiao YM, Chin PJ, Liu YL, et al. (2004). Genetic polymorphisms of the HCR gene and a genomic segment in close proximity to HLA-C are associated with patients with psoriasis in Taiwan. Br. J. Dermatol. 150: 1104-1111.
http://dx.doi.org/10.1111/j.1365-2133.2004.05972.x
PMid:15214895
Chang YT, Liu HN, Shiao YM, Lin MW, et al. (2005). A study of PSORS1C1 gene polymorphisms in Chinese patients with psoriasis. Br. J. Dermatol. 153: 90-96.
http://dx.doi.org/10.1111/j.1365-2133.2005.06570.x
PMid:16029332
Chang YT, Chou CT, Shiao YM, Lin MW, et al. (2006). Psoriasis vulgaris in Chinese individuals is associated with PSORS1C3 and CDSN genes. Br. J. Dermatol. 155: 663-669.
http://dx.doi.org/10.1111/j.1365-2133.2006.07420.x
PMid:16965413
Duffin KC, Chandran V, Gladman DD, Krueger GG, et al. (2008). Genetics of psoriasis and psoriatic arthritis: update and future direction. J. Rheumatol. 35: 1449-1453.
PMid:18609743 PMCid:2724000
Fan X, Yang S, Sun LD, Liang YH, et al. (2007). Comparison of clinical features of HLA-Cw*0602-positive and -negative psoriasis patients in a Han Chinese population. Acta Derm. Venereol. 87: 335-340.
http://dx.doi.org/10.2340/00015555-0253
PMid:17598037
Farber EM, Nall ML and Watson W (1974). Natural history of psoriasis in 61 twin pairs. Arch Dermatol. 109: 207-211.
http://dx.doi.org/10.1001/archderm.1974.01630020023005
PMid:4814926
Fojtíková M, Stolfa J, Novota P, Cejkova P, et al. (2009). HLA-Cw*06 class I region rather than MICA is associated with psoriatic arthritis in Czech population. Rheumatol. Int. 29: 1293-1299.
http://dx.doi.org/10.1007/s00296-009-0847-1
PMid:19184033
Gonzalez S, Martinez-Borra J, Torre-Alonso JC, Gonzalez-Roces S, et al. (1999). The MICA-A9 triplet repeat polymorphism in the transmembrane region confers additional susceptibility to the development of psoriatic arthritis and is independent of the association of Cw*0602 in psoriasis. Arthritis Rheum. 42: 1010-1016.
http://dx.doi.org/10.1002/1529-0131(199905)42:5<1010::AID-ANR21>3.0.CO;2-H
Griffiths CE and Barker JN (2007). Pathogenesis and clinical features of psoriasis. Lancet 370: 263-271.
http://dx.doi.org/10.1016/S0140-6736(07)61128-3
Gudjonsson JE, Karason A, Antonsdottir A, Runarsdottir EH, et al. (2003). Psoriasis patients who are homozygous for the HLA-Cw*0602 allele have a 2.5-fold increased risk of developing psoriasis compared with Cw6 heterozygotes. Br. J. Dermatol. 148: 233-235.
http://dx.doi.org/10.1046/j.1365-2133.2003.05115.x
PMid:12588373
Gudjonsson JE, Johnston A, Sigmundsdottir H and Valdimarsson H (2004). Immunopathogenic mechanisms in psoriasis. Clin. Exp. Immunol. 135: 1-8.
http://dx.doi.org/10.1111/j.1365-2249.2004.02310.x
PMid:14678257 PMCid:1808928
Higgins JP and Thompson SG (2002). Quantifying heterogeneity in a meta-analysis. Stat. Med. 21: 1539-1558.
http://dx.doi.org/10.1002/sim.1186
PMid:12111919
Holm SJ, Sanchez F, Carlen LM, Mallbris L, et al. (2005a). HLA-Cw*0602 associates more strongly to psoriasis in the Swedish population than variants of the novel 6p21.3 gene PSORS1C3. Acta Derm. Venereol. 85: 2-8.
http://dx.doi.org/10.1080/00015550410023527
Holm SJ, Sakuraba K, Mallbris L, Wolk K, et al. (2005b). Distinct HLA-C/KIR genotype profile associates with guttate psoriasis. J. Invest. Dermatol. 125: 721-730.
http://dx.doi.org/10.1111/j.0022-202X.2005.23879.x
PMid:16185272
Jobim M, Jobim LF, Salim PH, Cestari TF, et al. (2008). A study of the killer cell immunoglobulin-like receptor gene KIR2DS1 in a Caucasoid Brazilian population with Psoriasis vulgaris. Tissue Antigens 72: 392-396.
http://dx.doi.org/10.1111/j.1399-0039.2008.01096.x
PMid:18643961
Liu Y, Helms C, Liao W, Zaba LC, et al. (2008). A genome-wide association study of psoriasis and psoriatic arthritis identifies new disease loci. PLoS Genet. 4: e1000041.
http://dx.doi.org/10.1371/journal.pgen.1000041
PMid:18369459 PMCid:2274885
Lowes MA, Bowcock AM and Krueger JG (2007). Pathogenesis and therapy of psoriasis. Nature 445: 866-873.
http://dx.doi.org/10.1038/nature05663
PMid:17314973
Luft FC (2005). Light shed on the common skin scourge, psoriasis. J. Mol. Med. 83: 933-934.
http://dx.doi.org/10.1007/s00109-005-0724-7
PMid:16323032
Luszczek W, Kubicka W, Cislo M, Nockowski P, et al. (2003). Strong association of HLA-Cw6 allele with juvenile psoriasis in Polish patients. Immunol. Lett. 85: 59-64.
http://dx.doi.org/10.1016/S0165-2478(02)00212-2
Mallbris L, Wolk K, Sanchez F and Stahle M (2009). HLA-Cw*0602 associates with a twofold higher prevalence of positive streptococcal throat swab at the onset of psoriasis: a case control study. BMC Dermatol. 9: 5.
http://dx.doi.org/10.1186/1471-5945-9-5
PMid:19480679 PMCid:2696405
Martínez-Borra J, Gonzalez S, Santos-Juanes J, Sanchez del RJ, et al. (2003). Psoriasis vulgaris and psoriatic arthritis share a 100 kb susceptibility region telomeric to HLA-C. Rheumatology 42: 1089-1092.
http://dx.doi.org/10.1093/rheumatology/keg304
PMid:12730526
Martínez-Borra J, Brautbar C, Gonzalez S, Enk CD, et al. (2005). The region of 150 kb telometic to HLA-C is associated with psoriasis in the Jewish population. J. Invest. Dermatol. 125: 928-932.
PMid:16297191
Nair RP, Stuart PE, Nistor I, Hiremagalore R, et al. (2006). Sequence and haplotype analysis supports HLA-C as the psoriasis susceptibility 1 gene. Am. J. Hum. Genet. 78: 827-851.
http://dx.doi.org/10.1086/503821
PMid:16642438
Naldi L and Mercuri SR (2010). Epidemiology of comorbidities in psoriasis. Dermatol. Ther. 23: 114-118.
http://dx.doi.org/10.1111/j.1529-8019.2010.01304.x
PMid:20415817
Nestle FO, Kaplan DH and Barker J (2009). Psoriasis. N. Engl. J. Med. 361: 496-509.
http://dx.doi.org/10.1056/NEJMra0804595
PMid:19641206
Peters JL, Sutton AJ, Jones DR, Abrams KR, et al. (2006). Comparison of two methods to detect publication bias in meta-analysis. JAMA 295: 676-680.
http://dx.doi.org/10.1001/jama.295.6.676
PMid:16467236
Płoski R, Luszczek W, Kuśnierczyk P, Nockowski P, et al. (2006). A role for KIR gene variants other than KIR2DS1 in conferring susceptibility to psoriasis. Hum. Immunol. 67: 521-526.
http://dx.doi.org/10.1016/j.humimm.2006.04.001
PMid:16829306
Rani R, Narayan R, Fernandez-Vina MA and Stastny P (1998). Role of HLA-B and C alleles in development of psoriasis in patients from North India. Tissue Antigens 51: 618-622.
http://dx.doi.org/10.1111/j.1399-0039.1998.tb03004.x
PMid:9694354
Raychaudhuri SP and Gross J (2000a). A comparative study of pediatric onset psoriasis with adult onset psoriasis. Pediatr. Dermatol. 17: 174-178.
http://dx.doi.org/10.1046/j.1525-1470.2000.01746.x
PMid:10886746
Raychaudhuri SP and Gross J (2000b). Psoriasis risk factors: role of lifestyle practices. Cutis 66: 348-352.
PMid:11107520
Raychaudhuri SP and Farber EM (2001). The prevalence of psoriasis in the world. J. Eur. Acad. Dermatol. Venereol. 15: 16-17.
http://dx.doi.org/10.1046/j.1468-3083.2001.00192.x
PMid:11451313
Reveille JD and Williams FM (2006). Infection and musculoskeletal conditions: Rheumatologic complications of HIV infection. Best Pract. Res. Clin. Rheumatol. 20: 1159-1179.
http://dx.doi.org/10.1016/j.berh.2006.08.015
Sánchez F, Holm SJ, Mallbris L, O’Brien KP, et al. (2004). STG does not associate with psoriasis in the Swedish population. Exp. Dermatol. 13: 413-418.
http://dx.doi.org/10.1111/j.0906-6705.2004.00170.x
PMid:15217361
Schon MP and Boehncke WH (2005). Psoriasis. N. Engl. J. Med. 352: 1899-1912.
http://dx.doi.org/10.1056/NEJMra041320
PMid:15872205
Szczerkowska-Dobosz A (2005). Human leukocyte antigens as psoriasis inheritance and susceptibility markers. Arch. Immunol. Ther. Exp. 53: 428-433.
Valdimarsson H (2007). The genetic basis of psoriasis. Clin. Dermatol. 25: 563-567.
http://dx.doi.org/10.1016/j.clindermatol.2007.08.010
PMid:18021893
Vandenbroucke JP, von Elm E, Altman DG, Gotzsche PC, et al. (2007). Strengthening the Reporting of Observational Studies in Epidemiology (STROBE): explanation and elaboration. Epidemiology 18: 805-835.
http://dx.doi.org/10.1097/EDE.0b013e3181577511
PMid:18049195
Watson W, Cann HM, Farber EM and Nall ML (1972). The genetics of psoriasis. Arch. Dermatol. 105: 197-207.
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PMid:5060862
Zhang XJ, Zhang AP, Yang S, Gao M, et al. (2003). Association of HLA class I alleles with Psoriasis vulgaris in southeastern Chinese Hans. J. Dermatol. Sci. 33: 1-6.
http://dx.doi.org/10.1016/S0923-1811(03)00157-9
Zintzaras E and Ioannidis JP (2005). Heterogeneity testing in meta-analysis of genome searches. Genet. Epidemiol. 28: 123-137.
http://dx.doi.org/10.1002/gepi.20048
PMid:15593093