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A. A. S. Reis, Silva, D. M., Curado, M. P., and da Cruz, A. D., Involvement of CYP1A1, GST, 72TP53 polymorphisms in the pathogenesis of thyroid nodules, vol. 9, pp. 2222-2229, 2010.
Agundez JA (2004). Cytochrome P450 gene polymorphism and cancer. Curr. Drug Metab. 5: 211-224. PMid:15180491   Almeida PS, Manoel WJ, Reis AA, Silva ER, et al. (2008). TP53 codon 72 polymorphism in adult soft tissue sarcomas. Genet. Mol. Res. 7: 1344-1352. PMid:19065769   Aral C, Çaglayan S, Ösizik G, Massoumilary S, et al. (2007). The association of P53 codon 72 polymorphism with thyroid cancer in Turkish patients. Marmara Med. J. 20: 1-5.   Boltze C, Roessner A, Landt O, Szibor R, et al. (2002). Homozygous proline at codon 72 of p53 as a potential risk factor favoring the development of undifferentiated thyroid carcinoma. Int. J. Oncol. 21: 1151-1154. PMid:12370767   Bozina N, Bradamante V and Lovric M (2009). Genetic polymorphism of metabolic enzymes P450 (CYP) as a susceptibility factor for drug response, toxicity, and cancer risk. Arh. Hig. Rada Toksikol. 60: 217-242. PMid:19581216   Bufalo NE, Leite JL, Guilhen AC, Morari EC, et al. (2006). Smoking and susceptibility to thyroid cancer: an inverse association with CYP1A1 allelic variants. Endocr. Relat. Cancer 13: 1185-1193. PMid:17158763   Chen RH, Chang CT, Wang TY, Huang WL, et al. (2008). p53 codon 72 proline/arginine polymorphism and autoimmune thyroid diseases. J. Clin. Lab. Anal. 22: 321-326. PMid:18803266   Dean DS and Gharib H (2008). Epidemiology of thyroid nodules. Best Pract. Res. Clin. Endocrinol. Metab. 22: 901-911. PMid:19041821   Gaspar J, Rodrigues S, Gil OM, Manita I, et al. (2004). Combined effects of glutathione S-transferase polymorphisms and thyroid cancer risk. Cancer Genet. Cytogenet. 151: 60-67. PMid:15120911   Gonçalves AJ, Carvalho LH, Serdeira K, Nakai MY, et al. (2007). Comparative analysis of the prevalence of the glutathione S-transferase (GST) system in malignant and benign thyroid tumor cells. São Paulo Med. J. 125: 289-291. PMid:18094897   Hegedüs L (2004). Clinical practice. The thyroid nodule. N. Engl. J. Med. 351: 1764-1771. PMid:15496625   Hegedüs L, Bonnema SJ and Bennedbaek FN (2003). Management of simple nodular goiter: current status and future perspectives. Endocr. Rev. 24: 102-132. PMid:12588812   Hernández A, Céspedes W, Xamena N, Surrallés J, et al. (2003). Glutathione S-transferase polymorphisms in thyroid cancer patients. Cancer Lett. 190: 37-44.   Ho T, Zhao C, Zheng R, Liu Z, et al. (2006). Glutathione S-transferase polymorphisms and risk of differentiated thyroid carcinomas: a case-control analysis. Arch. Otolaryngol. Head Neck Surg. 132: 756-761. PMid:16847185   Lemos MC, Coutinho E, Gomes L, Carrilho F, et al. (2008). Combined GSTM1 and GSTT1 null genotypes are associated with a lower risk of papillary thyroid cancer. J. Endocrinol. Invest. 31: 542-545. PMid:18591888   Masson LF, Sharp L, Cotton SC and Little J (2005). Cytochrome P-450 1A1 gene polymorphisms and risk of breast cancer: A HuGE review. Am. J. Epidemiol. 161: 901-915. PMid:15870154   Mazzaferri EL (2006). Managing small thyroid cancer. JAMA 295: 2179-2182.   Morari EC, Leite JL, Granja F, da Assumpcao LV, et al. (2002). The null genotype of glutathione s-transferase M1 and T1 locus increases the risk for thyroid cancer. Cancer Epidemiol. Biomarkers Prev. 11: 1485-1488. PMid:12433731   Nebert DW, McKinnon RA and Puga A (1996). Human drug-metabolizing enzyme polymorphisms: effects on risk of toxicity and cancer. DNA Cell Biol. 15: 273-280. PMid:8639263   Rossini A, Rapozo DC, Amorim LM, Macedo JM, et al. (2002). Frequencies of GSTM1, GSTT1, and GSTP1 polymorphisms in a Brazilian population. Genet. Mol. Res. 1: 233-240. PMid:14963830   Siraj AK, Ibrahim M, Al-Rasheed M, Abubaker J, et al. (2008). Polymorphisms of selected xenobiotic genes contribute to the development of papillary thyroid cancer susceptibility in Middle Eastern population. B.M.C. Med. Genet. 9: 61. PMid:18601742 PMCid:2492854   Song N, Tan W, Xing D and Lin D (2001). CYP 1A1 polymorphism and risk of lung cancer in relation to tobacco smoking: a case-control study in China. Carcinogenesis 22: 11-16. PMid:11159735   Sourvinos G, Rizos E and Spandidos DA (2001). p53 Codon 72 polymorphism is linked to the development and not the progression of benign and malignant laryngeal tumours. Oral Oncol. 37: 572-578.   Stankov K, Landi S, Gioia-Patricola L, Bonora E, et al. (2006). GSTT1 and M1 polymorphisms in Hürthle thyroid cancer patients. Cancer Lett. 240: 76-82. PMid:16427734   Sweeney C, Farrow DC, Schwartz SM, Eaton DL, et al. (2000). Glutathione S-transferase M1, T1, and P1 polymorphisms as risk factors for renal cell carcinoma: a case-control study. Cancer Epidemiol. Biomarkers Prev. 9: 449-454. PMid:10794492