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2016
M. P. de Morais, Curado, R. F., Silva, K. S. Fe, Moura, K. K. V. O., and Arruda, J. T., Male idiopathic infertility and the TP53 polymorphism in codon 72, vol. 15, no. 4, p. -, 2016.
Conflicts of interestThe authors declare no conflict of interest.ACKNOWLEDGMENTSResearch supported by funding provided by PUC-GO University. REFERENCESAllan DJ, Harmon BV, Roberts SA, et al (1992). Spermatogonial apoptosis has three morphologically recognizable phases and shows no circadian rhythm during normal spermatogenesis in the rat. Cell Prolif. 25: 241-250. http://dx.doi.org/10.1111/j.1365-2184.1992.tb01399.x Beumer TL, Roepers-Gajadien HL, Gademan IS, van Buul PP, et al (1998). The role of the tumor suppressor p53 in spermatogenesis. Cell Death Differ. 5: 669-677. http://dx.doi.org/10.1038/sj.cdd.4400396 Bojesen SE, Nordestgaard BG, et al (2008). The common germline Arg72Pro polymorphism of p53 and increased longevity in humans. Cell Cycle 7: 158-163. http://dx.doi.org/10.4161/cc.7.2.5249 Brenna SMF, Silva IDCG, Zeferino LC, Pereira J, et al (2004). Prevalence of codon 72 p53 polymorphism in Brazilian women with cervix cancer. Genet. Mol. 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