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Found 11 results
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2016
I. R. Costa, Mascarenhas, R. S., Corrêa, J. F., Oliveira, F. R. B., Fernandes, S. G., Souza, F. O., Reis, G. Mdos, A. Júnior, deP. R., Rodrigues, D. A., Martins, J. V. M., Silva, K. S. Fe, Moura, K. K. V. O., Costa, I. R., Mascarenhas, R. S., Corrêa, J. F., Oliveira, F. R. B., Fernandes, S. G., Souza, F. O., Reis, G. Mdos, A. Júnior, deP. R., Rodrigues, D. A., Martins, J. V. M., Silva, K. S. Fe, and Moura, K. K. V. O., Analysis of the prevalence of polymorphisms in the glutathione S transferase gene (GST) in cataract patients from Goiânia, vol. 15, p. -, 2016.
I. R. Costa, Mascarenhas, R. S., Corrêa, J. F., Oliveira, F. R. B., Fernandes, S. G., Souza, F. O., Reis, G. Mdos, A. Júnior, deP. R., Rodrigues, D. A., Martins, J. V. M., Silva, K. S. Fe, Moura, K. K. V. O., Costa, I. R., Mascarenhas, R. S., Corrêa, J. F., Oliveira, F. R. B., Fernandes, S. G., Souza, F. O., Reis, G. Mdos, A. Júnior, deP. R., Rodrigues, D. A., Martins, J. V. M., Silva, K. S. Fe, and Moura, K. K. V. O., Analysis of the prevalence of polymorphisms in the glutathione S transferase gene (GST) in cataract patients from Goiânia, vol. 15, p. -, 2016.
M. P. de Morais, Curado, R. F., Silva, K. S. Fe, Moura, K. K. V. O., and Arruda, J. T., Male idiopathic infertility and the TP53 polymorphism in codon 72, vol. 15, no. 4, p. -, 2016.
Conflicts of interestThe authors declare no conflict of interest.ACKNOWLEDGMENTSResearch supported by funding provided by PUC-GO University. REFERENCESAllan DJ, Harmon BV, Roberts SA, et al (1992). Spermatogonial apoptosis has three morphologically recognizable phases and shows no circadian rhythm during normal spermatogenesis in the rat. Cell Prolif. 25: 241-250. http://dx.doi.org/10.1111/j.1365-2184.1992.tb01399.x Beumer TL, Roepers-Gajadien HL, Gademan IS, van Buul PP, et al (1998). The role of the tumor suppressor p53 in spermatogenesis. Cell Death Differ. 5: 669-677. http://dx.doi.org/10.1038/sj.cdd.4400396 Bojesen SE, Nordestgaard BG, et al (2008). The common germline Arg72Pro polymorphism of p53 and increased longevity in humans. Cell Cycle 7: 158-163. http://dx.doi.org/10.4161/cc.7.2.5249 Brenna SMF, Silva IDCG, Zeferino LC, Pereira J, et al (2004). Prevalence of codon 72 p53 polymorphism in Brazilian women with cervix cancer. Genet. Mol. Biol. 27: 496-499. http://dx.doi.org/10.1590/S1415-47572004000400005 Chowdhury AK, Steinberger E, et al (1964). A quantitative study of the effect of heat on germinal epithelium of rat testes. Am. J. Anat. 115: 509-524. http://dx.doi.org/10.1002/aja.1001150307 Churchman ML, Roig I, Jasin M, Keeney S, et al (2011). Expression of arf tumor suppressor in spermatogonia facilitates meiotic progression in male germ cells. PLoS Genet. 7: e1002157. http://dx.doi.org/10.1371/journal.pgen.1002157 Dumont P, Leu JI, Della Pietra AC3rdGeorgeDL, et al (2003). The codon 72 polymorphic variants of p53 have markedly different apoptotic potential. Nat. Genet. 33: 357-365. http://dx.doi.org/10.1038/ng1093 Fouchécourt S, Livera G, Messiaen S, Fumel B, et al (2016). Apoptosis of Sertoli cells after conditional ablation of murine double minute 2 (Mdm2) gene is p53-dependent and results in male sterility. Cell Death Differ. 23: 521-530. http://dx.doi.org/10.1038/cdd.2015.120 Huckins C, et al (1978). The morphology and kinetics of spermatogonial degeneration in normal adult rats: an analysis using a simplified classification of the germinal epithelium. Anat. Rec. 190: 905-926. http://dx.doi.org/10.1002/ar.1091900410 Jobling MA, Tyler-Smith C, et al (2003). The human Y chromosome: an evolutionary marker comes of age. Nat. Rev. Genet. 4: 598-612. http://dx.doi.org/10.1038/nrg1124 Kerr JB, et al (1992). Spontaneous degeneration of germ cells in normal rat testis: assessment of cell types and frequency during the spermatogenic cycle. J. Reprod. Fertil. 95: 825-830. http://dx.doi.org/10.1530/jrf.0.0950825 Lane DP, et al (1992). Cancer. p53, guardian of the genome. Nature 358: 15-16. http://dx.doi.org/10.1038/358015a0 Lattuada D, Viganò P, Somigliana E, Abbiati A, et al (2004). Analysis of the codon 72 polymorphism of the TP53 gene in patients with endometriosis. Mol. Hum. Reprod. 10: 651-654. http://dx.doi.org/10.1093/molehr/gah093 Li C, Chen K, Liu Z, Wang LE, et al (2008). Polymorphisms of TP53 Arg72Pro, but not p73 G4C14>A4TA4 and p21 Ser31Arg, contribute to risk of cutaneous melanoma. J. Invest. Dermatol. 128: 1585-1588. http://dx.doi.org/10.1038/sj.jid.5701186 Lin HY, Huang CH, Wu WJ, Chang LC, et al (2008). TP53 codon 72 Gene Polymorphism Paradox in Associated with Various Carcinoma Incidences, Invasiveness and Chemotherapy Responses. Int. J. Biomed. Sci. 4: 248-254. Lin YC, Yao PL, Richburg JH, et al (2010). FasL gene-deficient mice display a limited disruption in spermatogenesis and inhibition of mono-(2-ethylhexyl) phthalate-induced germ cell apoptosis. Toxicol. Sci. 114: 335-345. http://dx.doi.org/10.1093/toxsci/kfq015 Lu NX, Xia YK, Gu AH, Liang J, et al (2007). Lack of association between polymorphisms in p53 gene and spermatogenetic failure in a Chinese population. Andrologia 39: 223-228. http://dx.doi.org/10.1111/j.1439-0272.2007.00790.x Matzuk MM, Lamb DJ, et al (2008). The biology of infertility: research advances and clinical challenges. Nat. Med. 14: 1197-1213. http://dx.doi.org/10.1038/nm.f.1895 Murphy ME, et al (2006). Polymorphic variants in the p53 pathway. Cell Death Differ. 13: 916-920. http://dx.doi.org/10.1038/sj.cdd.4401907 Ohta H, Aizawa S, Nishimune Y, et al (2003). Functional analysis of the p53 gene in apoptosis induced by heat stress or loss of stem cell factor signaling in mouse male germ cells. Biol. Reprod. 68: 2249-2254. http://dx.doi.org/10.1095/biolreprod.102.014779 Olesen C, Hansen C, Bendsen E, Byskov AG, et al (2001). Identification of human candidate genes for male infertility by digital differential display. Mol. Hum. Reprod. 7: 11-20. http://dx.doi.org/10.1093/molehr/7.1.11 Pelúzio MCG, Volp ACP, Queiroz IC, Brito CJ, et al (2006). As proteínas supressoras em neoplasias malignas - Conhecendo seu papel. Rev. Bras. Nutr. Clín. 21: 233-238. Print CG, Loveland KL, et al (2000). Germ cell suicide: new insights into apoptosis during spermatogenesis. BioEssays 22: 423-430. http://dx.doi.org/10.1002/(SICI)1521-1878(200005)22:5<423::AID-BIES4>3.0.CO;2-0 Rotter V, Schwartz D, Almon E, Goldfinger N, et al (1993). Mice with reduced levels of p53 protein exhibit the testicular giant-cell degenerative syndrome. Proc. Natl. Acad. Sci. USA 90: 9075-9079. http://dx.doi.org/10.1073/pnas.90.19.9075 Schwartz D, Goldfinger N, Rotter V, et al (1993). Expression of p53 protein in spermatogenesis is confined to the tetraploid pachytene primary spermatocytes. Oncogene 8: 1487-1494. Shikone T, Billig H, Hsueh AJ, et al (1994). Experimentally induced cryptorchidism increases apoptosis in rat testis. Biol. Reprod. 51: 865-872. http://dx.doi.org/10.1095/biolreprod51.5.865 Show MD, Hill CM, Anway MD, Wright WW, et al (2008). Phosphorylation of mitogen-activated protein kinase 8 (MAPK8) is associated with germ cell apoptosis and redistribution of the Bcl2-modifying factor (BMF). J. Androl. 29: 338-344. http://dx.doi.org/10.2164/jandrol.107.003558 Siddique M, Sabapathy K, et al (2006). Trp53-dependent DNA-repair is affected by the codon 72 polymorphism. Oncogene 25: 3489-3500. http://dx.doi.org/10.1038/sj.onc.1209405 Stankiewicz P, Lupski JR, et al (2002). Genome architecture, rearrangements and genomic disorders. Trends Genet. 18: 74-82. http://dx.doi.org/10.1016/S0168-9525(02)02592-1 Tada M, Furuuchi K, Kaneda M, Matsumoto J, et al (2001). Inactivate the remaining p53 allele or the alternate p73? Preferential selection of the Arg72 polymorphism in cancers with recessive p53 mutants but not transdominant mutants. Carcinogenesis 22: 515-517. http://dx.doi.org/10.1093/carcin/22.3.515 WHO (1999). World Health Organization Laboratory manual for the examination of human semen and sperm-cervical mucus interaction. 4th edn. Cambridge University Press, Cambridge.