Interleukin-17

IL-17 rs2275913 genetic variation contributes to the development of gastric cancer in a Chinese population

B. L. Xu, Li, Y. T., Dong, S. X., Qi, J., Feng, H. M., Zi, L., Yang, D. Y., Xu, B. L., Li, Y. T., Dong, S. X., Qi, J., Feng, H. M., Zi, L., and Yang, D. Y., “IL-17 rs2275913 genetic variation contributes to the development of gastric cancer in a Chinese population”, vol. 15, p. -, 2016.

The purpose of this hospital-based case-control study was to assess whether the interleukin (IL)-17 rs2275913 genetic variation can influence susceptibility to gastric cancer. Samples from a total of 202 gastric cancer patients and 237 controls were collected from the Linyi People’s Hospital between March 2013 and March 2015. The IL-17 rs2275913 gene polymorphism was identified by polymerase chain reaction and restriction fragment length polymorphism.

Interleukin-17 enhances the removal of respiratory syncytial virus in mice by promoting neutrophil migration and reducing interferon-gamma expression

G. Zhang, Zhou, K. F., Lu, Z. H., Zhang, G., Zhou, K. F., Lu, Z. H., Zhang, G., Zhou, K. F., and Lu, Z. H., “Interleukin-17 enhances the removal of respiratory syncytial virus in mice by promoting neutrophil migration and reducing interferon-gamma expression”, vol. 15, p. -, 2016.

The aim of this study was to observe the effect of interleukin (IL)-17 on early immune response and inflammation in the lungs of respiratory syncytial virus (RSV)-infected mice. Specific pathogen-free BALB/c mice were randomly assigned to control, RSV-infected, RSV-infected with phosphate-buffered saline, and RSV-infected + IL-17 intervention groups. The RSV infection model was set up by nasal mucosa immunization. The intervention group was provided with restructured IL-17 (intranasal).

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