Inflammatory cytokines

IL-8 -251T/A polymorphism is associated with susceptibility to acute pancreatitis

X. B. Bao, Ma, Z., Gu, J. B., Wang, X. Q., Li, H. G., and Wang, W. Y., IL-8 -251T/A polymorphism is associated with susceptibility to acute pancreatitis, vol. 14, pp. 1508-1514, 2015.

We conducted a case-control study to clarify the asso­ciations between inflammatory cytokine, including interleukin (IL)-1b, IL-6, IL-8, and IL-10, polymorphisms and risk of acute pancreatitis. Genotyping analyses of IL-1β+3954 C/T (rs1143634), IL-1β-511 C/T (rs16944), IL-6 -174 G/C (rs1800795), IL-6 -634 C/G (rs1800796), IL-8 -251T/A (rs4073), IL-10 -1082A/G (rs1800896), and IL-10 -819C/T (rs1800871) were conducted using polymerase chain reaction-restriction fragment length of polymorphism.

Association between IL-1β, IL-8, and IL-10 polymorphisms and risk of acute pancreatitis

D. Li, Li, J., Wang, L., and Zhang, Q., Association between IL-1β, IL-8, and IL-10 polymorphisms and risk of acute pancreatitis, vol. 14, pp. 6635-6641, 2015.

We assessed the possible correlation between genetic polymorphisms in interleukin (IL)-1β, IL-8, and IL-10 and risk of acute pancreatitis. Polymorphisms of IL-1β+3954C/T (rs1143634), IL-1β-511C/T (rs16944), IL-8 -251T/A (rs4073), IL-10 -1082A/G (rs1800896), and IL-10 -819C/T (rs1800871) were assessed by polymerase chain reaction-restriction fragment length polymorphism.

Xuemaitong granules attenuate carotid atherosclerosis by decreasing the expression of CD14+CD16+ monocytes, IL-6, TNF-α, and hsCRP

Q. Zhang, Qian, G., and Ding, Z., Xuemaitong granules attenuate carotid atherosclerosis by decreasing the expression of CD14+CD16+ monocytes, IL-6, TNF-α, and hsCRP, vol. 13, pp. 7519-7527, 2014.

Carotid atherosclerosis (CAS) has been extensively studied because its position can be easily observed. Our objective was to investigate the effects of Xuemaitong granules on the generation and activation of CD14+CD16+ monocytes on the inflammatory reaction in CAS patients. In this study, 22 male apolipoprotein E (apoE)-deficient mice were fed a high-fat diet for 13 weeks.

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