Brain natriuretic peptide

Transcriptional analysis of atrial and ventricular muscles from rats

Y. Zhi, Cao, Z., Li, Q. H., Li, X. L., Sun, Y., Zhang, T., Zhang, Q., Zhi, Y., Cao, Z., Li, Q. H., Li, X. L., Sun, Y., Zhang, T., Zhang, Q., Zhi, Y., Cao, Z., Li, Q. H., Li, X. L., Sun, Y., Zhang, T., and Zhang, Q., Transcriptional analysis of atrial and ventricular muscles from rats, vol. 15, p. -, 2016.

Previous studies have used microarray technology to explore gene expression differences between the atrium and the ventricle. However, selection criteria for the differentially expressed genes (DEGs) based only on either the fold change or the P value in these studies. Here, we aim to further identify the DEGs by setting a P value threshold of 2, which may yield more specific gene expression differences between the atrium and the ventricle.

Protective effect of lyophilized recombinant human brain natriuretic peptide on renal ischemia/reperfusion injury in mice

X. Cao, Xia, H. Y., Zhang, T., Qi, L. C., Zhang, B. Y., Cui, R., Chen, X., Zhao, Y. R., and Li, X. Q., Protective effect of lyophilized recombinant human brain natriuretic peptide on renal ischemia/reperfusion injury in mice, vol. 14, pp. 13300-13311, 2015.

Brain natriuretic peptide (BNP) has a protective effect on acute injury of the heart, brain, and lung. However, its role in acute kidney injury (AKI) remains unclear. The aim of this study was to investigate the effect of lyophilized recombinant human BNP (lrh-BNP) on AKI and the underlying molecular mechanisms. An experimental model for AKI was established using an ischemia/reperfusion (I/R) procedure. Healthy adult BALB/c mice were randomized to the sham, I/R, and lrh-BNP-treated post-I/R (BNP + I/R) groups.

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